C5a is important in the tubulointerstitial component of experimental immune complex glomerulonephritis

Clinical and Experimental Immunology

Volumn 130, Issue 1, 2002, Pages 43-48

  Welch, T R ^a,b,e   Frenzke, M ^a,b   Witte, D ^a,c   Davis III, A E ^d  

^a CINCINNATI CHILDREN S HOSPITAL RESEARCH FOUNDATION   (United States)

^b UNIVERSITY OF CINCINNATI   (United States)

^c UNIVERSITY OF CINCINNATI COLLEGE OF MEDICINE   (United States)

^d CENTER FOR BLOOD RESEARCH   (United States)

^e STATE UNIVERSITY OF NEW YORK   (United States)

Author keywords

C5a receptor; Complement; Glomerulonephritis; Renal tubule

Indexed keywords

APOFERRITIN; COMPLEMENT COMPONENT C5A; COMPLEMENT COMPONENT C5A RECEPTOR; LIPOPOLYSACCHARIDE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; ATROPHY; CELL COUNT; CELL INFILTRATION; CONTROLLED STUDY; EXTRACELLULAR MATRIX; GENE EXPRESSION; GLOMERULONEPHRITIS; IN SITU HYBRIDIZATION; KIDNEY PARENCHYMA; KIDNEY TUBULE EPITHELIUM; KNOCKOUT MOUSE; LEYDIG CELL; MOUSE; MOUSE STRAIN; NONHUMAN; PRIORITY JOURNAL; PROTEIN FUNCTION; STRAIN DIFFERENCE; WILD TYPE;

ANIMALS; ANIMALS, CONGENIC; ANTIGENS, CD; APOFERRITINS; APOPTOSIS; ATROPHY; COMPLEMENT ACTIVATION; COMPLEMENT C3; EPITHELIAL CELLS; GENE EXPRESSION REGULATION; GLOMERULONEPHRITIS; HEMATURIA; HORSES; IMMUNE COMPLEX DISEASES; IN SITU HYBRIDIZATION; KIDNEY GLOMERULUS; KIDNEY TUBULES; LIPOPOLYSACCHARIDES; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MODELS, ANIMAL; PROTEINURIA; RECEPTOR, ANAPHYLATOXIN C5A; RECEPTORS, COMPLEMENT;

EID: 0036378385     PISSN: 00099104     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1365-2249.2002.01957.x     Document Type: Article

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