|
Volumn 88, Issue 2, 2002, Pages 335-344
|
Quenching of intracellular ROS generation as a mechanism for oleate-induced reduction of endothelial activation and early atherogenesis
a a a a a a a a a |
Author keywords
Adhesion molecules; Glutathione; Oleate; Reactive oxygen species; Redox status
|
Indexed keywords
CATALASE;
COLONY STIMULATING FACTOR 1;
CYTOKINE;
ENZYME;
FATTY ACID;
GLUTATHIONE;
GLUTATHIONE PEROXIDASE;
GLUTATHIONE REDUCTASE;
GLUTATHIONE TRANSFERASE;
INTERLEUKIN 1ALPHA;
LIPOPOLYSACCHARIDE;
OLEIC ACID;
REACTIVE OXYGEN METABOLITE;
SUPEROXIDE;
SUPEROXIDE DISMUTASE;
TUMOR NECROSIS FACTOR ALPHA;
VASCULAR CELL ADHESION MOLECULE 1;
ANIMAL CELL;
AORTA;
ARTICLE;
ATHEROGENESIS;
CATTLE;
CELL ACTIVATION;
CELL FREE SYSTEM;
CELL LEVEL;
CELL STIMULATION;
CELL TYPE;
CELL VIABILITY;
CONTROLLED STUDY;
DEPLETION;
DRUG EFFECT;
DRUG EXPOSURE;
ENDOTHELIUM CELL;
ENZYME ACTIVITY;
ENZYME ASSAY;
HUMAN;
HUMAN CELL;
LIPID COMPOSITION;
METABOLIC INHIBITION;
MICROVASCULATURE;
NONHUMAN;
PRIORITY JOURNAL;
PROTECTION;
PROTEIN ANALYSIS;
PROTEIN EXPRESSION;
PROTEIN SECRETION;
PROTEIN SYNTHESIS INHIBITION;
QUANTITATIVE ANALYSIS;
SAPHENOUS VEIN;
STIMULUS RESPONSE;
UMBILICAL VEIN;
VASCULAR ENDOTHELIUM;
|
EID: 0036336462
PISSN: 03406245
EISSN: None
Source Type: Journal
DOI: 10.1055/s-0037-1613207 Document Type: Article |
Times cited : (76)
|
References (58)
|