Effects of halothane and enflurane anesthesia on sympathetic β-adrenoreceptor-mediated pulmonary vasodilation in chronically instrumented dogs

Anesthesiology

Volumn 97, Issue 2, 2002, Pages 478-487

  Sato, Kosei ^b   Seki, Sumihiko ^b   Murray, Paul A ^a  

^a LERNER RESEARCH INSTITUTE   (United States)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

15 HYDROXY 11ALPHA,9ALPHA EPOXYMETHANOPROSTA 5,13 DIENOIC ACID; BETA ADRENERGIC RECEPTOR; BUCLADESINE; CYCLIC AMP; ENFLURANE; HALOTHANE; ISOPRENALINE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BLOOD PRESSURE; CONTROLLED STUDY; DOG; HEMODYNAMICS; HYPOTENSION; LUNG VASCULAR RESISTANCE; LUNG VASODILATION; NONHUMAN; PRIORITY JOURNAL; RECEPTOR DENSITY; SIGNAL TRANSDUCTION; VASODILATATION;

ANESTHESIA, INHALATION; ANESTHETICS, INHALATION; ANIMALS; BLOOD GLUCOSE; BUCLADESINE; DOGS; DRUG SYNERGISM; ENFLURANE; HALOTHANE; HEMODYNAMIC PROCESSES; ISOFLURANE; PULMONARY CIRCULATION; RECEPTORS, ADRENERGIC, BETA; VASODILATION;

EID: 0036330094     PISSN: 00033022     EISSN: None     Source Type: Journal    
DOI: 10.1097/00000542-200208000-00027     Document Type: Article

References (28)

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3. Isoflurane anesthesia attenuates endothelium-dependent pulmonary vasorelaxation by inhibiting the synergistic interaction between nitric oxide and prostacyclin
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