Mechanism of doxorubicin-induced cell death and expression profile analysis

Biotechnology Letters

Volumn 24, Issue 14, 2002, Pages 1147-1151

  Lee, Seongeun ^a   Baek, Myungin ^a   Kim, Hae Yeong ^b,c   Ha, Ji Hong ^c   Jeoung, Doo Il ^a  

^a In2Gen Co Ltd   (South Korea)

^b Kyung Hee University   (South Korea)

^c Kyungpook National University   (South Korea)

Author keywords

Apoptosis; CDNA microarray; Doxorubicin; Gastric cancer cells

Indexed keywords

CASPASE; COMPLEMENTARY DNA; DOXORUBICIN; NICOTINAMIDE ADENINE DINUCLEOTIDE ADENOSINE DIPHOSPHATE RIBOSYLTRANSFERASE;

APOPTOSIS; ARTICLE; CANCER CELL; CANCER CELL CULTURE; CELL DEATH; CONTROLLED STUDY; DNA MICROARRAY; ENZYME ACTIVATION; ENZYME ACTIVITY; GENE EXPRESSION PROFILING; GENE IDENTIFICATION; HUMAN; HUMAN CELL; PHENOTYPE; STOMACH CANCER;

EID: 0036325542     PISSN: 01415492     EISSN: None     Source Type: Journal    
DOI: 10.1023/A:1016174800956     Document Type: Article

References (8)

1. Anthracycline in the treatment of cancer. An overview
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3. Monitoring the expression profiles of doxorubicin-induced and doxorubcin-resistant cancer cells by cDNA microarray
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5. Involvement of transcriptional repressor ATF3 in acceleration of caspase protease activation during DNA damaging agent-induced apoptosis
6. p53 activates the CD95 gene in response to DNA damage by anticancer drugs
7. A DNA fragment from Streptomyces peucetius activates biosynthesis of actinorhodin in Strepto. Lividans and daunomycin in the original strain
8. Apoptosis in the pathogenesis and treatment of disease