A role for endogenous glucocorticoids in wound repair

EMBO Reports

Volumn 3, Issue 6, 2002, Pages 575-582

  Grose, Richard ^a   Werner, Silke ^a   Kessler, Daniela ^a   Tuckermann, Jan ^a   Huggel, Katharina ^a   Durka, Silke ^a   Reichardt, Holger M ^a   Werner, Sabine ^a  

^a ETH ZURICH   (Switzerland)

Author keywords

[No Author keywords available]

Indexed keywords

COLLAGEN; DNA; GLUCOCORTICOID; GLUCOCORTICOID RECEPTOR; MUTANT PROTEIN; FIBRONECTIN; GROWTH PROMOTOR; INTERLEUKIN 1; SCLEROPROTEIN; TRANSFORMING GROWTH FACTOR BETA;

ANIMAL CELL; ANIMAL TISSUE; ARTICLE; CELL DENSITY; CELL MIGRATION; CELL PROLIFERATION; CONTROLLED STUDY; DNA BINDING; FIBROBLAST; GRANULATION TISSUE; IN VITRO STUDY; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; REGULATORY MECHANISM; WOUND HEALING; ANIMAL; GENETICS; METABOLISM; PHYSIOLOGY;

ANIMALS; EXTRACELLULAR MATRIX PROTEINS; FIBROBLASTS; FIBRONECTINS; GLUCOCORTICOIDS; GRANULATION TISSUE; GROWTH SUBSTANCES; INTERLEUKIN-1; MICE; RECEPTORS, GLUCOCORTICOID; TRANSFORMING GROWTH FACTOR BETA; WOUND HEALING;

EID: 0036306763     PISSN: 1469221X     EISSN: None     Source Type: Journal    
DOI: 10.1093/embo-reports/kvf119     Document Type: Article

References (19)

1. Glucocorticoid-regulated gene expression during cutaneous wound repair
2. Production of a tissue-like structure by contraction of collagen lattices by human fibroblasts of different proliferative potential in vitro
3. Macrophages and fibroblasts express embryonic fibronectins during cutaneous wound healing
4. Cell proliferation and cell cycle progression are not impaired in fibroblasts and ES cells lacking c-Fos
5. Impaired mechanical stability, migration and contractile capacity in vimentin-deficient fibroblasts
6. Transforming growth factors β1, β2 and β3 and their receptors are differentially regulated during normal and impaired wound healing
7. A crucial role of β1 integrins for keratinocyte migration in vitro and during cutaneous wound repair
8. Differential regulation of pro-inflammatory cytokines during wound healing in normal and glucocorticoid-treated mice
9. Fibroblasts in mechanically stressed collagen lattices assume a "synthetic" phenotype
10. Glucocorticoids selectively inhibit the transcription of the interleukin 1β gene and decrease the stability of interleukin 1β mRNA
11. The role of the macrophage in wound repair. A study with hydrocortisone and antimacrophage serum
12. Restraint stress slows cutaneous wound healing in mice
13. DNA binding of the glucocorticoid receptor is not essential for survival
14. Repression of inflammatory responses in the absence of DNA binding by the glucocorticoid receptor
15. Quantitative studies of the growth of mouse embryo cells in culture and their development into established lines
16. The DNA binding-independent function of the glucocorticoid receptor mediates repression of AP-1-dependent genes in skin
17. Large induction of keratinocyte growth factor in the dermis during wound healing
18. The function of KGF in morphogenesis of epithelium and reepithelialization of wounds
19. Tissue repair and fibrosis