Pathogenesis of postinfarction free wall rupture [1]

International Journal of Cardiology

Volumn 84, Issue 1, 2002, Pages 91-92

  Beranek, Jiri T ^a  

^a NONE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

FAT;

APOPTOSIS; BLOOD PRESSURE; HEART INFARCTION; HEART LEFT VENTRICLE WALL; HEART MUSCLE CELL; HEART RUPTURE; HEART TAMPONADE; HEMODYNAMICS; HUMAN; LETTER; NEUTROPHIL; PATHOGENESIS; PRIORITY JOURNAL;

APOPTOSIS; HEART RUPTURE, POST-INFARCTION; HUMANS; MYOCARDIUM;

EID: 0036298602     PISSN: 01675273     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0167-5273(02)00142-0     Document Type: Letter

References (10)

1. Survival following self-limited left ventricular free wall rupture during myocardial infarction. Management differences between patients with or without pseudoaneurysm formation
2. Why primary angioplasty is less offensive to the myocardium compared with thrombolysis for acute myocardial infarction
3. Science: Problems, aims, responsibilities
4. Biomechanical properties of reperfused transmural myocardial infarcts in rabbits during the first week after infarction. Implications for left ventricular rupture
5. Alterations of the myocardial skeletal framework in acute myocardial infarction with and without ventricular rupture. A preliminary report
6. On the "cause" of tuberculosis
7. Pathogenesis of transverse midventricular disruption
8. Ischemic heart disease
9. The floating heart or the heart too fat to sink: Analysis of 55 necropsy patients