Pathogenesis of transverse midventricular disruption [8]

International Journal of Cardiology

Volumn 81, Issue 2-3, 2001, Pages 291-292

  Beranek, Jiri T ^a  

^a NONE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

APOPTOSIS; ARTERIAL PRESSURE; BLEEDING; CONNECTIVE TISSUE; EOSINOPHILIA; HEART LEFT VENTRICLE; HEART MUSCLE; HEART MUSCLE NECROSIS; HEART RUPTURE; HISTOPATHOLOGY; LETTER; LONGITUDINAL STUDY; MAMMAL; MITRAL VALVE REPLACEMENT; MYOCARDIAL DISEASE; PATHOGENESIS; PRIORITY JOURNAL; TRANSVERSE MIDVENTRICULAR DISRUPTION;

HEART INJURIES; HEART VALVE PROSTHESIS IMPLANTATION; HEART VENTRICLES; HUMANS; POSTOPERATIVE COMPLICATIONS;

EID: 0035664222     PISSN: 01675273     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0167-5273(01)00549-6     Document Type: Letter

References (8)

1. Transverse midventricular disruption after mitral valve replacement
2. Avoidance of transverse midventricular disruption following mitral valve replacement
3. Biomechanical properties of reperfused transmural myocardial infarcts in rabbits during the first week after infarction
4. Pathogenesis of heart fibrosis in systemic sclerosis
5. Why primary angioplasty is less offensive to the myocardium compared with thrombolysis for acute myocardial infarction
6. Quick disposal of dead cardiomyocytes: An ultimate proof of their apoptosis
7. The Vineberg legacy
8. Insulin-like growth factor-1 induces Mdm2 and down-regulates p53, attenuating the myocyte renin-angiotensin system and stretch-mediated apoptosis