A place for TACE

Gut

Volumn 51, Issue 1, 2002, Pages 5-6

  Van Deventer, S J H ^a  

^a ACADEMIC MEDICAL CENTER   (Netherlands)

Author keywords

[No Author keywords available]

Indexed keywords

TUMOR NECROSIS FACTOR ALPHA; TUMOR NECROSIS FACTOR ALPHA CONVERTING ENZYME;

ANIMAL EXPERIMENT; ANIMAL MODEL; COLITIS; CYTOKINE PRODUCTION; CYTOKINE RELEASE; ENTERITIS; ENZYME ACTIVITY; HUMAN; INFLAMMATION; MONONUCLEAR CELL; MOUSE; NONHUMAN; PRIORITY JOURNAL; SHORT SURVEY; T LYMPHOCYTE;

ADAM PROTEINS; COLONIC DISEASES, FUNCTIONAL; ENZYME INHIBITORS; HUMANS; METALLOENDOPEPTIDASES; RECEPTORS, TUMOR NECROSIS FACTOR;

EID: 0036288636     PISSN: 00175749     EISSN: None     Source Type: Journal    
DOI: 10.1136/gut.51.1.5     Document Type: Short Survey

References (12)

1. Tuberculosis associated with infliximab, a tumor necrosis factor alpha-neutralizing agent
2. A murine transmembrane tumor necrosis factor (TNF) transgene induces arthritis by cooperative p55/p75 TNF receptor signaling
3. Cloning of a disintegrin metalloproteinase that processes precursor tumour-necrosis factor-alpha
4. A metalloproteinase disintegrin that releases tumour-necrosis factor-alpha from cells
5. Tumour necrosis factor α converting enzyme (TACE) activity in the colonic mucosa of patients with inflammatory bowel disease
6. The effect of a metalloproteinase inhibitor (GI5402) on tumor necrosis factor-alpha (TNF-alpha) and TNF-alpha receptors during human endotoxemia
7. Transmembrane TNF is sufficient to induce localized tissue toxicity and chronic inflammatory arthritis in transgenic mice
8. The metalloproteinase inhibitor GI5402 inhibits endotoxin-induced soluble CD27 and CD16 release in healthy humans
9. Paradoxical effects of a synthetic metalloproteinase inhibitor that blocks both p55 and p75 TNF receptor shedding and TNF alpha processing in RA synovial membrane cell cultures
10. The effect of an inhibitor of matrix metalloproteinases on colonic inflammation in a trinitrobenzenesulphonic acid rat model of inflammatory bowel disease
11. Imbalance of stromelysin-1 and TIMP-1 in the mucosal lesions of children with inflammatory bowel disease
12. Infliximab induces apoptosis in monocytes from patients with chronic active crohn's disease by using a caspase-dependent pathway