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Volumn 87, Issue 2, 2002, Pages 115-116
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Novel genes, proteins, and inherited disorders of iron overload: Iron metabolism is less boring than thought
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Author keywords
[No Author keywords available]
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Indexed keywords
FERRITIN;
HFE PROTEIN;
IRON;
IRON REGULATORY FACTOR;
TRANSFERRIN;
EDITORIAL;
FERRITIN BLOOD LEVEL;
GENE EXPRESSION;
GENE MUTATION;
GENETIC DISORDER;
HOMEOSTASIS;
HUMAN;
INBORN ERROR OF METABOLISM;
IRON METABOLISM;
IRON OVERLOAD;
PATHOGENESIS;
ADULT;
AGE OF ONSET;
AMINO ACID SUBSTITUTION;
ANIMALS;
ANTIMICROBIAL CATIONIC PEPTIDES;
CATION TRANSPORT PROTEINS;
CHILD;
DNA-BINDING PROTEINS;
EUROPE;
GENE EXPRESSION REGULATION;
GENETIC HETEROGENEITY;
HEMOCHROMATOSIS;
HISTOCOMPATIBILITY ANTIGENS CLASS I;
HLA ANTIGENS;
HOMEOSTASIS;
HUMANS;
IRON;
IRON OVERLOAD;
MAMMALS;
MEMBRANE PROTEINS;
MICE;
MICE, KNOCKOUT;
MULTIGENE FAMILY;
MUTATION, MISSENSE;
POINT MUTATION;
RECEPTORS, TRANSFERRIN;
TRANSCRIPTION FACTORS;
UPSTREAM STIMULATORY FACTORS;
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EID: 0036181423
PISSN: 03906078
EISSN: None
Source Type: Journal
DOI: None Document Type: Editorial |
Times cited : (13)
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References (27)
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