Reduced lipolysis as possible cause for greater weight gain in subjects with the Pro12Ala polymorphism in PPARγ2? [4]

Diabetologia

Volumn 45, Issue 1, 2002, Pages 152-153

  Stumvoll, M ^a  

^a UNIVERSITY HOSPITAL TÜBINGEN   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

PEROXISOME PROLIFERATOR ACTIVATED RECEPTOR GAMMA;

GENETIC POLYMORPHISM; GENOTYPE; GLUCOSE HOMEOSTASIS; GLUCOSE TOLERANCE; HUMAN; INSULIN SENSITIVITY; LETTER; LIPOLYSIS; NON INSULIN DEPENDENT DIABETES MELLITUS; OBESITY; PRIORITY JOURNAL; RISK FACTOR; WEIGHT GAIN;

ADULT; ALANINE; AMINO ACID SUBSTITUTION; BLOOD GLUCOSE; BODY MASS INDEX; FATTY ACIDS, NONESTERIFIED; FEMALE; HUMANS; INSULIN; LIPOLYSIS; MALE; POLYMORPHISM, GENETIC; PROLINE; RECEPTORS, CYTOPLASMIC AND NUCLEAR; TRANSCRIPTION FACTORS; WEIGHT GAIN;

EID: 0036162822     PISSN: 0012186X     EISSN: None     Source Type: Journal    
DOI: 10.1007/s125-002-8257-y     Document Type: Letter

References (8)

1. 2 gene on long-term weight change in Finnish non-diabetic subjects
2. The Pro12Ala polymorphism in the peroxisome proliferator activated receptor g2 gene is associated with increased antilipolytic insulin sensitivity
3. A novel use of the hyperinsulinemic-euglycemic clamp technique to measure insulin sensivity of systemic lipolysis
4. The common PPAR gamma Pro12Ala polymorphism is associated with decreased risk of type 2 diabetes
5. Lipoatrophy revisited
6. PPAR gamma mediates high-fat diet-induced adipocyte hypertrophy and insulin resistance
7. A Pro12Ala substitution in PPARgamma2 associated with decreased receptor activity, lower body mass index and improved insulin sensitivity
8. Activators of peroxisome proliferator-activated receptor gamma have depot-specific effects on human preadipocyte differentiation