Functional implications of mutations in the human renal outer medullary potassium channel (ROMK2) identified in bartter syndrome

Pflugers Archiv European Journal of Physiology

Volumn 443, Issue 3, 2002, Pages 466-472

  Starremans, Patrick G J F ^a   Van der Kemp, Annemiete W C M ^a   Knoers, Nine V A M ^a   Van den Heuvel, Lambertus P W J ^a   Bindels, René J M ^a  

^a RADBOUD UNIVERSITY MEDICAL CENTER   (Netherlands)

Author keywords

Bartter syndrome; Kidney; Renal electrolyte transport; ROMK

Indexed keywords

BARIUM ION; COMPLEMENTARY RNA; POTASSIUM CHANNEL; SODIUM CHLORIDE;

ANIMAL CELL; ARTICLE; BARTTER SYNDROME; CELL MEMBRANE; ELECTROLYTE TRANSPORT; GENE MUTATION; HENLE LOOP; HUMAN; ION CURRENT; KIDNEY EPITHELIUM; NONHUMAN; PRIORITY JOURNAL; SODIUM ABSORPTION;

ANIMALS; BARTTER SYNDROME; HUMANS; IMMUNOHISTOCHEMISTRY; KIDNEY MEDULLA; MEMBRANE POTENTIALS; MUTAGENESIS; OOCYTES; PATCH-CLAMP TECHNIQUES; POTASSIUM CHANNELS; POTASSIUM CHANNELS, INWARDLY RECTIFYING; PROTEIN STRUCTURE, TERTIARY; WATER-ELECTROLYTE BALANCE; XENOPUS LAEVIS;

EID: 0036156486     PISSN: 00316768     EISSN: None     Source Type: Journal    
DOI: 10.1007/s004240100708     Document Type: Article

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