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Volumn 34, Issue 2, 2002, Pages 194-203
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Relationship between glucocorticoid-mediated early decrease of protein synthesis and the steady state decreases of glucocorticoid receptor and TGF-β activator protein
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Author keywords
Collagen synthesis; Dexamethasone; Glucocorticoid receptor DNA binding; Protein synthesis; TGF activator protein DNA binding
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Indexed keywords
COLLAGEN;
DEXAMETHASONE;
DNA;
GLUCOCORTICOID;
GLUCOCORTICOID RECEPTOR;
IMMUNOGLOBULIN ENHANCER BINDING PROTEIN;
PROTEIN;
TRANSCRIPTION FACTOR SP1;
TRANSFORMING GROWTH FACTOR BETA ACTIVATOR PROTEIN;
UNCLASSIFIED DRUG;
ANIMAL CELL;
ANIMAL TISSUE;
ARTICLE;
CELL CULTURE;
COLLAGEN SYNTHESIS;
CONTROLLED STUDY;
DERMIS;
FETUS;
IN VIVO STUDY;
NEWBORN;
NONHUMAN;
PROTEIN DNA BINDING;
PROTEIN SYNTHESIS;
PROTEIN SYNTHESIS INHIBITION;
RAT;
SKIN FIBROBLAST;
STEADY STATE;
TISSUE REPAIR;
ANIMALS;
CELLS, CULTURED;
COLLAGEN;
DEXAMETHASONE;
DNA;
DOSE-RESPONSE RELATIONSHIP, DRUG;
FIBROBLASTS;
GLUCOCORTICOIDS;
OLIGODEOXYRIBONUCLEOTIDES;
OLIGODEOXYRIBONUCLEOTIDES, ANTISENSE;
PROTEIN BIOSYNTHESIS;
RATS;
RATS, SPRAGUE-DAWLEY;
RECEPTORS, GLUCOCORTICOID;
SKIN;
TRANSFORMING GROWTH FACTOR BETA;
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EID: 0036156127
PISSN: 13572725
EISSN: None
Source Type: Journal
DOI: 10.1016/S1357-2725(01)00095-4 Document Type: Article |
Times cited : (7)
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References (34)
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