Hepatic encephalopathy

Neurologic Clinics

Volumn 20, Issue 1, 2002, Pages 241-246

  Lockwood, Alan H ^a,b  

^a UNIVERSITY AT BUFFALO   (United States)

^b VA WESTERN NEW YORK HEALTHCARE SYSTEM   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

BRAIN DYSFUNCTION; DIAGNOSTIC IMAGING; HEPATIC ENCEPHALOPATHY; HUMAN; LIVER CIRRHOSIS; NOMENCLATURE; NUCLEAR MAGNETIC RESONANCE IMAGING; POSITRON EMISSION TOMOGRAPHY; PRIORITY JOURNAL; QUALITY OF LIFE; REVIEW;

EID: 0036154885     PISSN: 07338619     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0733-8619(03)00061-6     Document Type: Review

References (17)

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3. Subclinical hepatic encephalopathy impairs daily functioning
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5. Positron-emission tomographic localization of abnormalities of brain metabolism in patients with minimal hepatic encephalopathy
6. Neuropsychological test deficits correlate with altered cerebral glucose metabolism in patients with non-alcoholic cirrhosis
7. An image of the brain in patients with liver disease
8. Cerebral ammonia metabolism in patients with severe liver disease and minimal hepatic encephalopathy
9. The effect of lactulose on psychomotor performance tests in alcoholic cirrhotics without overt hepatic encephalopathy
10. Lactitol and lactulose for the treatment of subclinical hepatic encephalopathy in cirrhotic patients. A randomised, cross-over study
11. The diagnosis of subclinical hepatic encephalopathy in patients with cirrhosis using neuropsychological tests and automated electroencephalogram analysis
12. Subclinical hepatic encephalopathy: Detection, prevalence, and relationship to nitrogen metabolism
13. Neuropsychological aspects of portal-systemic encephalopathy
14. Latent portasystemic encephalopathy: I. Nature of cerebral functional defects and their effect on fitness to drive
15. Fitness to drive in patients with cirrhosis and portal-systemic shunting: A pilot study evaluating driving performance
16. Nonalcoholic cirrhosis associated with neuropsychological dysfunction in the absence of overt evidence of hepatic encephalopathy