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Volumn 99, Issue 5, 2002, Pages 629-634
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Transformation by inorganic arsenic compounds of normal Syrian hamster embryo cells into a neoplastic state in which they become anchorage-independent and cause tumors in newborn hamsters
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Author keywords
Anchorage independence; Arsenic; DNA hypomethylation; Neoplastic transformation; Senescence; Syrian hamster embryo cell
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Indexed keywords
ARSENATE SODIUM;
ARSENIC DERIVATIVE;
CYTOSINE;
GUANINE;
INORGANIC COMPOUND;
MESSENGER RNA;
ANCHORAGE INDEPENDENT GROWTH;
ANIMAL CELL;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
ARTICLE;
CANCER CELL CULTURE;
CARCINOGENESIS;
CARCINOGENICITY;
CELL GROWTH;
CELL IMMORTALIZATION;
CONTROLLED STUDY;
DIPLOIDY;
DNA METHYLATION;
EMBRYO CELL;
FEMALE;
GENE AMPLIFICATION;
GENE EXPRESSION;
MALIGNANT TRANSFORMATION;
NEWBORN;
NONHUMAN;
NUCLEOTIDE SEQUENCE;
ONCOGENE C MYC;
ONCOGENE RAS;
PHENOTYPE;
PRIORITY JOURNAL;
SENESCENCE;
SYRIAN HAMSTER;
ANIMALS;
ANIMALS, NEWBORN;
ARSENATES;
ARSENITES;
CARCINOGENS;
CELL TRANSFORMATION, NEOPLASTIC;
CELLS, CULTURED;
CRICETINAE;
DEOXYRIBONUCLEASE HPAII;
DNA METHYLATION;
EMBRYO;
GENE AMPLIFICATION;
GENES, RAS;
MESOCRICETUS;
NEOPLASM TRANSPLANTATION;
PROTO-ONCOGENE PROTEINS C-MYC;
RNA, MESSENGER;
SODIUM COMPOUNDS;
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EID: 0036105865
PISSN: 00207136
EISSN: None
Source Type: Journal
DOI: 10.1002/ijc.10407 Document Type: Article |
Times cited : (49)
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References (30)
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