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Low c-kit expression of cultured mast cells of mi/mi genotype may be involved in their defective responses to fibroblasts that express the ligand for c-kit
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Mutations at the mouse microphthalmia locus are associated with defects in a gene encoding a novel basic helix-loop-helix zipper protein
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Systematic method to obtain novel genes that are regulated by mi transcription factor (MITF): Impaired expression of granzyme B and tryptophan hydroxylase in mi/mi cultured mast cells
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Inhibitory effect of the transcription factor encoded by the mi mutant allele in cultured mast cells of mice
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Deficient transcription of mouse mast cell protease 4 gene in mutant mice of mi/mi genotype
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Loss of DNA binding ability of the transcription factor encoded by the mutant mi locus
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Regulation of mouse mast cell protease 6 gene expression by transcription factor encoded by the mi locus
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Abnormal expression of mouse mast cell protease 5 gene in cultured mast cells derived from mutant mi/mi mice
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Effect of a large deletion of the basic domain of mi transcription factor on differentiation of mast cells
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ce mutant mice of which MITF lacks the zipper domain
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Inhibitory effect of transcription factor encoded by mutant mi microphthalmia allele on transactivation of mouse mast cell protease 7 gene
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Ogihara H., Morii E., Kim D.K., Oboki K., Kitamura Y. Inhibitory effect of transcription factor encoded by mutant mi microphthalmia allele on transactivation of mouse mast cell protease 7 gene. Blood. 97:2001;645-651.
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Effect of the mi allele on mast cells, basophils, natural killer cells, and osteoclasts in C57Bl/6J mice
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Molecular basis of mouse microphthalmia (mi) mutations helps explain their developmental and phenotypic consequences
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The recessive phenotype displayed by a dominant negative microphthalmia-associated transcription factor mutant is a result of impaired nuclear localization potential
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Involvement of transcription factor encoded by the mi locus in the expression of c-kit receptor tyrosine kinase in cultured mast cell of mice
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