An unequal crossover event in RCCX modules of the human MHC resulting in the formation of a TNXB/TNXA hybrid and deletion of the CYP21A

Human Immunology

Volumn 63, Issue 8, 2002, Pages 683-689

  Jaatinen, Taina ^a   Chung, Erwin K ^b,c   Ruuskanen, Olli ^d   Lokki, Marja Liisa ^a  

^a FINNISH RED CROSS BLOOD SERVICE   (Finland)

^b THE RESEARCH INSTITUTE AT NATIONWIDE CHILDREN S HOSPITAL   (United States)

^c OHIO STATE UNIVERSITY   (United States)

^d TURKU UNIVERSITY HOSPITAL   (Finland)

Author keywords

C4; Deletion; MHC; Recombination; TNX

Indexed keywords

ARTICLE; CROSSING OVER; CYP21A GENE; CYP21B GENE; GENE; GENE DELIVERY SYSTEM; GENE DUPLICATION; GENETIC RECOMBINATION; HAPLOTYPE; HUMAN; JUVENILE RHEUMATOID ARTHRITIS; MAJOR HISTOCOMPATIBILITY COMPLEX; PRIORITY JOURNAL; RESTRICTION FRAGMENT LENGTH POLYMORPHISM; TNX GENE; TNXB GENE; BIOLOGICAL MODEL; CASE REPORT; GENE DELETION; GENETICS; HYBRIDIZATION; MALE;

CASE REPORT; COMPLEMENT 4B; CROSSING OVER (GENETICS); GENE DELETION; GENE DUPLICATION; HAPLOTYPES; HUMAN; HYBRIDIZATION, GENETIC; MAJOR HISTOCOMPATIBILITY COMPLEX; MALE; MODELS, GENETIC; PROTEINS; RECOMBINATION, GENETIC; STEROID 21-HYDROXYLASE; SUPPORT, NON-U.S. GOV'T; SUPPORT, U.S. GOV'T, P.H.S.; TENASCIN; COMPLEMENT C4B; CROSSING OVER, GENETIC; HUMANS;

COMPLEMENT COMPONENT C4B; PROTEIN; STEROID 21 MONOOXYGENASE; TENASCIN; TENASCIN X; TNXA PROTEIN, HUMAN;

EID: 0036020522     PISSN: 01988859     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0198-8859(02)00416-0     Document Type: Article

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