Na+/i- symporter activity requires a small and uncharged amino acid residue at position 395

Molecular Endocrinology

Volumn 16, Issue 8, 2002, Pages 1893-1902

  Dohán, Orsolya ^a   Veronica Gavrielides M ^a   Ginter, Christopher ^a   Mario Amzel, L ^b   Carrasco, Nancy ^a  

^a ALBERT EINSTEIN COLLEGE OF MEDICINE   (United States)

^b JOHNS HOPKINS UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

AMINO ACID; ASPARTIC ACID; COMPLEMENTARY DNA; GLUTAMIC ACID; GLYCINE; IODIDE; LYSINE; MEMBRANE PROTEIN; SODIUM ION; THYROID HORMONE; COTRANSPORTER; DNA; RECOMBINANT PROTEIN; SODIUM; SODIUM IODIDE SYMPORTER;

AMINO ACID SUBSTITUTION; ANIMAL CELL; ARTICLE; CONGENITAL HYPOTHYROIDISM; GENE MUTATION; ION TRANSPORT; NONHUMAN; PRIORITY JOURNAL; SITE DIRECTED MUTAGENESIS; STRUCTURE ACTIVITY RELATION; THYROID GLAND; ANIMAL; CELL STRAIN COS1; CHEMICAL STRUCTURE; CHEMISTRY; GENETIC TRANSFECTION; GENETICS; HUMAN; KINETICS; METABOLISM; NUCLEOTIDE SEQUENCE; PROTEIN SECONDARY STRUCTURE; RAT;

AMINO ACID SUBSTITUTION; AMINO ACIDS, NEUTRAL; ANIMAL; BASE SEQUENCE; COS CELLS; DNA; HUMAN; IODIDES; KINETICS; MODELS, MOLECULAR; MUTAGENESIS, SITE-DIRECTED; PROTEIN STRUCTURE, SECONDARY; RATS; RECOMBINANT PROTEINS; SODIUM; SUPPORT, U.S. GOV'T, P.H.S.; SYMPORTERS; TRANSFECTION; ANIMALS; HUMANS;

ANIMALIA;

EID: 0036020509     PISSN: 08888809     EISSN: None     Source Type: Journal    
DOI: 10.1210/me.2002-0071     Document Type: Article

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