Central 5-HT3 receptor stimulation by m-CPBG increases blood glucose in rats

Hormone and Metabolic Research

Volumn 34, Issue 2, 2002, Pages 55-61

  Carvalho, F ^a   Macedo D ^a   Bandeira, I ^a   Maldonado, I ^a   Salles, L ^a   Azevedo, M F ^a   Rocha Jr , M A ^a   Fregoneze, J B ^a   De Castro e Silva, Emilio ^a  

^a FEDERAL UNIVERSITY OF BAHIA   (Brazil)

Author keywords

Blood Glucose; Hyperglycemia; Serotonin; Stress

Indexed keywords

1 (3 CHLOROPHENYL)BIGUANIDE; GLUCOSE; ONDANSETRON; SEROTONIN; SEROTONIN 3 RECEPTOR; 1-(3-CHLOROPHENYL)BIGUANIDE; BIGUANIDE DERIVATIVE; SEROTONIN AGONIST; SEROTONIN ANTAGONIST; SEROTONIN RECEPTOR;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN THIRD VENTRICLE; CONTROLLED STUDY; DIET RESTRICTION; DRUG RECEPTOR BINDING; FOOD INTAKE; GLUCOSE BLOOD LEVEL; HYPERGLYCEMIA; HYPOTHALAMUS HYPOPHYSIS ADRENAL SYSTEM; IMMOBILIZATION STRESS; MALE; NONHUMAN; PRIORITY JOURNAL; RAT; SEROTONINERGIC SYSTEM; SYMPATHETIC TONE; ANIMAL; BLOOD; EXERCISE; FOOD; INTRACEREBROVENTRICULAR DRUG ADMINISTRATION; KINETICS; METABOLISM; PHYSIOLOGY; STRESS; WISTAR RAT;

ANIMALIA;

ANIMALS; BIGUANIDES; BLOOD GLUCOSE; FASTING; FOOD; INJECTIONS, INTRAVENTRICULAR; KINETICS; MALE; ONDANSETRON; RATS; RATS, WISTAR; RECEPTORS, SEROTONIN; RECEPTORS, SEROTONIN, 5-HT3; RESTRAINT, PHYSICAL; SEROTONIN AGONISTS; SEROTONIN ANTAGONISTS; STRESS;

EID: 0036009008     PISSN: 00185043     EISSN: None     Source Type: Journal    
DOI: 10.1055/s-2002-20525     Document Type: Article

References (29)

1. 1A and alpha-2 adrenergic receptors mediate the hyperglycemic and hypoinsulinemic effects of 8-hydroxy-2-(di-n-propylamino) tetralin in conscious rat
2. 2 receptor agonists DOI and α-methyl-5-HT on plasma glucose and insulin levels in the rat
3. 2 receptors are involved in the adrenal catecholamine-releasing and hyperglycemic effects of the 5-hydroxytryptamine indirect agonist d-fenfluramine in the conscious rat
4. Modulation of the hypothermia and hyperglycaemic effects of 8-OH-DPAT by α2-adrenoceptor antagonists
5. 2B/2C receptor agonist I-(3-chlorophenyl)piperazine on the plasma glucose levels of rats
6. 2B/2C receptors
7. The hypothalamus, intrinsic connections and outflow pathways to the endocrine system in relation to the control of feeding and metabolism
8. Hyperglycemic properties of serotonin receptor antagonists
9. Involvement of hypothalamic serotonin in activation of the sympathoadrenomedullary system and hypothalamo-pituitary-adrenocortical axis in male Wistar rats
10. Ganglionic transmission is a prerequisite for the adrenaline-releasing and hyperglycemic effects of 8-OH-DPAT
11. The concepts of stress and stress system disorders. Overview of physical and behavioral homeostasis
12. Physiopharmacological interactions between stress hormones and central serotonergic systems
13. A rapid and simple procedure for chronic cannulation of rat jugular vein
14. 3 receptor agonist
15. 4 receptor antagonists
16. Selective serotonin reuptake inhibitors fluoxetine and fluvoxamine induce hyperglycemia by different mechanisms
17. Role of central neural mechanisms in the regulation of hepatic glucose metabolism
18. Reciprocal influences of the ventromedial and lateral hypothalamic nuclei on blood glucose level and liver glycogen content
19. Central nervous system control of glycogenolysis and gluneogenesis in fed and fasted rat liver
20. 2 receptor agonist DOI
21. 3 receptor in CNS disorders
22. 3 receptors in neuronal cell lines
23. Role of central neural mechanisms in the regulation of hepatic glucose metabolism
24. 3 receptors and GABAergic neurons inhibiting acetylcholine release in rat enthorhinal cortex slices
25. 3 receptors mediate inhibition of acetylcholine release in cortical tissue
26. Central command neurons of the sympathetic nervous system: Basis of the fight-or-flight response
27. Serotonin and stress
28. Serotonin agonists cause parallel activation of sympathoadrenomedullary system and the hypothalamo-pituitary-adrenocortical axis in conscious rats
29. Closing the gap in prophylactic antiemetic therapy: Patient factors in calculating the emetogenic potential of chemotherapy