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Oncogene

Volumn 20, Issue 49 REV. IIS. 6, 2001, Pages 7136-7139

APL, a model disease for cancer therapies?

(2) De Thé, Hugues a Chelbi Alix, Mounira K b

a SAINT LOUIS HOSPITAL (France)

b INSTITUT ANDRÉ LWOFF (France)

Author keywords

Apoptosis; Arsenic; Differentiation; Leukemia; PML RAR ; Retinoic acid

Indexed keywords

ANTHRACYCLINE; ARSENIC; HORMONE RECEPTOR; RETINOIC ACID; RETINOIC ACID RECEPTOR; TELOMERASE; UBIQUITIN;

ACUTE MYELOBLASTIC LEUKEMIA; ANIMAL MODEL; APOPTOSIS; CANCER THERAPY; CARCINOGENESIS; CELL DIFFERENTIATION; CHROMOSOME TRANSLOCATION 15; DIMERIZATION; DNA BINDING; GENE REARRANGEMENT; LEUKEMIA REMISSION; MALIGNANT TRANSFORMATION; PRIORITY JOURNAL; REVIEW; TRANSCRIPTION REGULATION;

ANTINEOPLASTIC AGENTS; ARSENICALS; CELL AGING; CELL DIFFERENTIATION; CELL NUCLEUS; HUMANS; INCLUSION BODIES; LEUKEMIA, PROMYELOCYTIC, ACUTE; MULTIGENE FAMILY; NEOPLASM PROTEINS; ONCOGENE PROTEINS, FUSION; OXIDES; RECEPTORS, RETINOIC ACID; TRETINOIN;

ANIMALIA;

EID: 0035969096 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1204851 Document Type: Review

Times cited : (21)

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