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Volumn 69, Issue 10, 2001, Pages 1167-1179
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Are adhesion molecules involved in stress-induced changes in lymphocyte distribution?
b
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Author keywords
Adhesion molecules; Chronic stress; Glucocorticoids; Hypothalamic pituitary adrenal axis; Restraint
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Indexed keywords
BETA2 INTEGRIN;
CD11B ANTIGEN;
CELL ADHESION MOLECULE;
GLUCOCORTICOID;
INTEGRIN;
LYMPHOCYTE FUNCTION ASSOCIATED ANTIGEN 1;
UNCLASSIFIED DRUG;
ANIMAL CELL;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ARTICLE;
CELL COUNT;
CELLULAR DISTRIBUTION;
CONTROLLED STUDY;
CORTICOSTERONE BLOOD LEVEL;
HABITUATION;
HELPER CELL;
IMMOBILIZATION STRESS;
LYMPHOCYTE SUBPOPULATION;
MALE;
NATURAL KILLER CELL;
NONHUMAN;
RAT;
T LYMPHOCYTE;
TIME;
ACUTE DISEASE;
ADRENAL GLANDS;
ANIMALS;
ANTIGENS, CD18;
CELL COUNT;
CHRONIC DISEASE;
CORTICOSTERONE;
IMMUNOPHENOTYPING;
KILLER CELLS, NATURAL;
LYMPHOCYTE FUNCTION-ASSOCIATED ANTIGEN-1;
LYMPHOCYTE SUBSETS;
MACROPHAGE-1 ANTIGEN;
MALE;
ORGAN SIZE;
RATS;
RATS, SPRAGUE-DAWLEY;
RESTRAINT, PHYSICAL;
STRESS, PSYCHOLOGICAL;
T-LYMPHOCYTES, HELPER-INDUCER;
THYMUS GLAND;
TIME FACTORS;
UP-REGULATION;
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EID: 0035958623
PISSN: 00243205
EISSN: None
Source Type: Journal
DOI: 10.1016/S0024-3205(01)01200-0 Document Type: Article |
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Times cited : (36)
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References (40)
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