Prevalence of donor-specific anti-HLA antibodies during episodes of renal allograft rejection

Transplantation

Volumn 71, Issue 4, 2001, Pages 577-580

  Supon, Patrick ^a,b,c   Constantino, Don ^a,b,c   Hao, Ping ^a,b,c   Cagle, Linda ^a,b,c   Hahn, Amy ^a,b,c   Conti, David J ^a,b,c   Freed, Brian M ^a,b,c,d  

^a UNIVERSITY OF COLORADO   (United States)

^b ALBANY MEDICAL COLLEGE   (United States)

^c KUNMING MEDICAL UNIVERSITY   (China)

^d UNIVERSITY OF COLORADO   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CYCLOSPORIN; HLA ANTIBODY; HLA ANTIGEN CLASS 1; HLA ANTIGEN CLASS 2; IMMUNOGLOBULIN G ANTIBODY; IMMUNOSUPPRESSIVE AGENT;

ANTIBODY DETECTION; ANTIBODY SPECIFICITY; ARTICLE; CYTOMEGALOVIRUS INFECTION; DRUG INDUCED DISEASE; ENZYME LINKED IMMUNOSORBENT ASSAY; HUMAN; KIDNEY ALLOGRAFT REJECTION; KIDNEY GRAFT REJECTION; KIDNEY TRANSPLANTATION; MAJOR CLINICAL STUDY; ORGAN TRANSPLANTATION; PRIORITY JOURNAL;

EID: 0035956957     PISSN: 00411337     EISSN: None     Source Type: Journal    
DOI: 10.1097/00007890-200102270-00017     Document Type: Article

References (18)

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10. The presence of posttransplant HLA-specific IgG antibodies detected by enzymelinked immunosorbent assay correlates with specific rejection pathologies
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12. Donor-specific antibodies after transplantation by flow cytometry
13. Identification of patients at high risk of graft loss by pre- and posttransplant monitoring of anti-HLA class I IgG antibodies by immunosuorbent assay
14. Anti-HLA antibodies detected in posttransplant renal allograft recipient sera correlate with chronic rejection
15. HLA class I and II antibodies: Monitoring before and after kidney transplantation and their clinical relevance
16. Anti-HLA antibodies after solid organ transplantation
17. Induction of high affinity fibroblast growth factor receptor expression and proliferation in human endothelial cells by anti-HLA antibodies: A possible mechanism for transplant atherosclerosis
18. Alloantibody-mediated class I signal transduction in endothelial cells and smooth muscle cells: Enhancement by IFN-γ and TNF-α