The cyclic AMP response element in the Bcl-2 promoter confers inducibility by hypoxia in neuronal cells

Molecular Brain Research

Volumn 92, Issue 1-2, 2001, Pages 98-106

  Freeland, Karen ^a   Boxer, Linda M ^b   Latchman, David S ^a  

^a UNIVERSITY COLLEGE LONDON   (United Kingdom)

^b STANFORD UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Apoptosis; Bcl 2; CREB transcription factor; Gene regulation; Hypoxia

Indexed keywords

CYCLIC AMP RESPONSIVE ELEMENT BINDING PROTEIN; NERVE GROWTH FACTOR; PROTEIN BCL 2; TRANSCRIPTION FACTOR;

ANIMAL CELL; ARTICLE; BRAIN HYPOXIA; CONTROLLED STUDY; ENZYME ACTIVATION; ENZYME ACTIVITY; GENE EXPRESSION; GENE INDUCTION; NERVE CELL; NEUROPROTECTION; NONHUMAN; PHOSPHORYLATION; PRIORITY JOURNAL; PROMOTER REGION; RAT; STIMULUS RESPONSE;

ANIMALS; APOPTOSIS; BINDING SITES; BUCLADESINE; CELL HYPOXIA; CREB-BINDING PROTEIN; CYCLIC AMP; CYCLIC AMP RESPONSE ELEMENT-BINDING PROTEIN; FUNGAL PROTEINS; GENE EXPRESSION REGULATION; GENES, BCL-2; GENES, REPORTER; LUCIFERASES; NEOPLASM PROTEINS; NERVE GROWTH FACTOR; NERVE TISSUE PROTEINS; NEURONS; NUCLEAR PROTEINS; OXIDATIVE STRESS; PC12 CELLS; PHOSPHORYLATION; PROMOTER REGIONS (GENETICS); PROTEIN KINASE C; PROTEIN PROCESSING, POST-TRANSLATIONAL; PROTO-ONCOGENE PROTEINS C-BCL-2; RATS; RECOMBINANT FUSION PROTEINS; REGULATORY SEQUENCES, NUCLEIC ACID; SACCHAROMYCES CEREVISIAE PROTEINS; SECOND MESSENGER SYSTEMS; TRANS-ACTIVATORS; TRANSCRIPTION FACTORS; TRANSCRIPTION, GENETIC; TRANSFECTION;

EID: 0035882349     PISSN: 0169328X     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0169-328X(01)00158-9     Document Type: Article

References (24)

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