Temporal relationships between ceramide production, caspase activation and mitochondrial dysfunction in cell lines with varying sensitivity to anti-Fas-induced apoptosis

Biochemical Journal

Volumn 357, Issue 2, 2001, Pages 407-416

  Rodriguez Lafrasse, C ^a   Alphonse, G ^a   Broquet, P ^a   Aloy, M T ^a   Louisot, P ^a   Rousson, R ^a  

^a INSERM   (France)

Author keywords

Jurkat cells; SCC61 cells; Sphingolipid metabolism; SQ20B cells

Indexed keywords

BIOSYNTHESIS; DRUG DOSAGE; ENZYME KINETICS; TUMORS;

APOPTOSIS;

CELL MEMBRANES;

CASPASE; CASPASE 3; CASPASE 8; CERAMIDE; FAS ANTIBODY; GLUTATHIONE; NICOTINAMIDE ADENINE DINUCLEOTIDE ADENOSINE DIPHOSPHATE RIBOSYLTRANSFERASE;

APOPTOSIS; ARTICLE; CELL LEVEL; CONTROLLED STUDY; DEPLETION; ENZYME ACTIVATION; ENZYME INHIBITION; HUMAN; HUMAN CELL; LEUKEMIA CELL LINE; LIPID METABOLISM; MEMBRANE POTENTIAL; MITOCHONDRION; PRIORITY JOURNAL; SIGNAL TRANSDUCTION; TUMOR CELL LINE;

ANTIBODIES, MONOCLONAL; ANTIGENS, CD95; APOPTOSIS; CARCINOMA, SQUAMOUS CELL; CASPASE 3; CASPASE 8; CASPASE 9; CASPASES; CELL ADHESION; CERAMIDES; CYSTEINE PROTEINASE INHIBITORS; ENZYME ACTIVATION; GLUTATHIONE; HEAD AND NECK NEOPLASMS; HUMANS; INTRACELLULAR MEMBRANES; JURKAT CELLS; KINETICS; MEMBRANE POTENTIALS; MITOCHONDRIA; POLY(ADP-RIBOSE) POLYMERASES; TUMOR CELLS, CULTURED;

EID: 0035879052     PISSN: 02646021     EISSN: None     Source Type: Journal    
DOI: 10.1042/0264-6021:3570407     Document Type: Article

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