Fine specificity of anti-GQ1b antibodies and clinical features

Journal of the Neurological Sciences

Volumn 185, Issue 1, 2001, Pages 1-2

  Willison, Hugh J ^a  

^a SOUTHERN GENERAL HOSPITAL   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

AUTOANTIBODY; GANGLIOSIDE; GQ1B ANTIBODY; UNCLASSIFIED DRUG;

ANTIBODY SPECIFICITY; ATAXIA; CLINICAL FEATURE; EDITORIAL; GUILLAIN BARRE SYNDROME; HUMAN; OPHTHALMOPLEGIA; PRIORITY JOURNAL;

EID: 0035869621     PISSN: 0022510X     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0022-510X(01)00465-8     Document Type: Note

References (13)

1. An unusual variant of acute idiopathic polyneuritis (syndrome of ophthalmoplegia, ataxia and areflexia)
2. Serum IgG antibody to ganglioside GQ1b is a possible marker of Miller Fisher syndrome
3. Clinical correlate and fine specificity of anti-GQ1b antibodies in peripheral neuropathy
4. Fine specificity of anti-GQ1b antibodies and clinical features
5. Miller Fisher syndrome and other acute variants of Guillain-Barré syndrome
6. Ophthalmoplegic and lower cranial nerve variants merge into each other and into classical Guillain-Barré syndrome
7. The pathogenesis of Miller Fisher syndrome
8. Acute oropharyngeal palsy is associated with anti bodies to GQ1b and GT1a gangliosides
9. Is IgG anti-GT1a antibody associated with pharyngeal-cervical-brachial weakness or oropharyngeal palsy in Guillain-Barré syndrome?
10. Anti-ganglioside antibody and neuropathy: review of our research
11. A somatically mutated human anti-ganglioside antibody that induces experimental neuropathy in mice is encoded by the variable region heavy chain gene V1-18
12. Monospecific anti-GD1b and IgG is required to induce rabbit ataxic neuropathy
13. Ganglioside composition of the human cranial nerves, with special reference to pathophysiology of Miller Fisher syndrome