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Proceedings of the National Academy of Sciences of the United States of America

Volumn 98, Issue 12, 2001, Pages 6865-6870

Proinflammatory cytokines regulate human glucocorticoid receptor gene expression and lead to the accumulation of the dominant negative β isoform: A mechanism for the generation of glucocorticoid resistance

(4) Webster, Jeffrey C a,b Oakley, Robert H a,c Jewell, Christine M a Cidlowski, John A a

a NATIONAL INSTITUTE OF ENVIRONMENTAL HEALTH SCIENCES (United States)

b DUPONT COMPANY (United States)

c DUKE UNIVERSITY MEDICAL CENTER (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CYTOKINE; GLUCOCORTICOID RECEPTOR; GLUCOCORTICOID RECEPTOR ALPHA; GLUCOCORTICOID RECEPTOR BETA; ISOPROTEIN; MESSENGER RNA; TRANSCRIPTION FACTOR; TUMOR NECROSIS FACTOR; TUMOR NECROSIS FACTOR ALPHA; UNCLASSIFIED DRUG;

ARTICLE; BINDING SITE; CONTROLLED STUDY; GENE EXPRESSION; GENE TRANSLOCATION; GENETIC TRANSCRIPTION; HORMONE ACTION; HORMONE BINDING; HUMAN; HUMAN CELL; INFLAMMATION; LIGAND BINDING; LYMPHOID CELL; PRIORITY JOURNAL; PROTEIN DNA BINDING; PROTEIN EXPRESSION;

ANIMALS; COS CELLS; CYTOKINES; DRUG RESISTANCE; GENE EXPRESSION REGULATION; GLUCOCORTICOIDS; HELA CELLS; HUMANS; INTERLEUKIN-1; NF-KAPPA B; PROTEIN ISOFORMS; RECEPTORS, GLUCOCORTICOID; RNA, MESSENGER; TUMOR NECROSIS FACTOR-ALPHA;

EID: 0035810930 PISSN: 00278424 EISSN: None Source Type: Journal
DOI: 10.1073/pnas.121455098 Document Type: Article

Times cited : (433)

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