메뉴 건너뛰기




Volumn 96, Issue 1, 2001, Pages 17-22

Sepsis increases NOS-2 activity and decreases non-NOS-mediated acetylcholine-induced dilation in rat aorta

Author keywords

[No Author keywords available]

Indexed keywords

ACETYLCHOLINE; ISOENZYME; N(G) METHYLARGININE; NITRIC OXIDE SYNTHASE; PHENYLEPHRINE; SODIUM CHLORIDE;

EID: 0035746420     PISSN: 00224804     EISSN: None     Source Type: Journal    
DOI: 10.1006/jsre.2000.6056     Document Type: Article
Times cited : (11)

References (39)
  • 8
    • 0027511853 scopus 로고
    • The discovery of nitric oxide in the vessel wall: A unifying concept in the pathogenesis of sepsis
    • (1993) Arch. Surg. , vol.128 , pp. 396
    • Palmer, R.M.J.1
  • 28
    • 0028367655 scopus 로고
    • Effects of cytokines tumor necrosis factor a and interleukin 1b on endotoxin-mediated inhibition of endothelium-derived relaxing factor bioactivity and nitric oxide production in vascular endothelium
    • (1994) Shock , vol.1 , pp. 73
    • Myers, P.R.1    Parker, J.L.2    Tanner, M.A.3    Adams, H.R.4
  • 30
    • 0027467022 scopus 로고
    • Selective Inhibition of endothelium-dependent vasodilator capacity by Escherichia coli endotoxemia
    • (1993) Circ. Res. , vol.72 , pp. 539
    • Parker, J.L.1    Adams, H.R.2


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.