Gene-environment interaction in the expression of antioxidant status: A role for genes in the relationship between smoking and coronary disease

Arteriosclerosis, Thrombosis, and Vascular Biology

Volumn 21, Issue 7, 2001, Pages 1102-1103

  Hokanson, John E ^a  

^a UNIVERSITY OF COLORADO   (United States)

Author keywords

Antioxidants; Genetics; Smoking

Indexed keywords

ANTIOXIDANT;

ANTIOXIDANT ACTIVITY; ATHEROSCLEROSIS; CORONARY ARTERY DISEASE; EDITORIAL; ENVIRONMENTAL FACTOR; GENE EXPRESSION; GENE INTERACTION; GENETIC ANALYSIS; GENETIC SUSCEPTIBILITY; HUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; SMOKING;

EID: 0035721933     PISSN: 10795642     EISSN: None     Source Type: Journal    
DOI: 10.1161/hq0701.093718     Document Type: Editorial

References (7)

1. Coronary atherosclerosis and oxidative stress as reflected by autoantibodies against oxidized low-density lipoprotein and oxysterols
2. Circulating oxidized LDL is a useful marker for identifying patients with coronary artery disease
3. Genetic contributions to plasma total antioxidant activity
4. 55 genotype
5. Smoking is associated with reduced serum paraoxonase activity and concentration in patients with coronary artery disease
6. Tyrosyl radical generated by myeloperoxidase is a physiological catalyst for initiation of lipid peroxidation of low density lipoprotein
7. An Alu element in the myeloperoxidase promoter contains a composite SP-1 thyroid hormone-retinoic acid response element