Circulating leptin: Mediator or marker of the neuroendocrinological stress response?

Critical Care Medicine

Volumn 29, Issue 12, 2001, Pages 2397-2398

  Stryjewski, G ^a   Dalton, H J ^a  

^a GEORGE WASHINGTON UNIVERSITY SCHOOL OF MEDICINE AND HEALTH SCIENCES   (United States)

Author keywords

Adipocyte; Cardiopulmonary by pass; Cortisol; Leptin; Pediatrics; Sepsis; Shock

Indexed keywords

BIOCHEMICAL MARKER; HYDROCORTISONE; LEPTIN;

CARDIOPULMONARY BYPASS; EDITORIAL; HORMONAL REGULATION; HORMONE BLOOD LEVEL; HORMONE RELEASE; HUMAN; HYDROCORTISONE BLOOD LEVEL; MEDIATOR RELEASE; NEUROENDOCRINOLOGY; PRIORITY JOURNAL; STRESS;

EID: 0035674779     PISSN: 00903493     EISSN: None     Source Type: Journal    
DOI: 10.1097/00003246-200112000-00032     Document Type: Editorial

References (9)

1. Leptin: Physiology and pathophysiology
2. Gain in body fat is inversely related to the nocturnal rise in serum leptin levels in young females
3. Glucocorticoid effects on the diurnal rhythm of circulating leptin levels
4. Evidence for a novel peripheral action of leptin as a metabolic signal to the adrenal gland: Leptin inhibits cortisol release directly
5. Circulating leptin and the perioperative neuroendocrinological stress response after pediatric cardiac surgery
6. Leptin and the perioperative neuroendocrinological stress response
7. Plasma leptin levels are increased in survivors of acute sepsis: Associated loss of diurnal rhythm in cortisol and leptin secretion
8. Reduced leptin levels in starvation increased susceptibility to endotoxic shock
9. Leptin is an endogenous protective protein against the toxicity exerted by tumor necrosis factor