Resveratrol inhibits TNFα-induced endothelial cell activation

Therapie

Volumn 56, Issue 5, 2001, Pages 613-616

  Bertelli, A A E ^a   Baccalini, R ^a   Battaglia, E ^a   Falchi, M ^a   Ferrero, M E ^a  

^a UNIVERSITY OF MILAN   (Italy)

Author keywords

Adhesion molecule; Endothelium; Inflammation; Resveratrol; Vascular permeability

Indexed keywords

ANTIINFLAMMATORY AGENT; RESVERATROL; TUMOR NECROSIS FACTOR ALPHA; VASCULAR CELL ADHESION MOLECULE 1;

ANIMAL MODEL; ANIMAL TISSUE; ANTIINFLAMMATORY ACTIVITY; BLOOD VESSEL PERMEABILITY; CELL ADHESION; CONFERENCE PAPER; CONTROLLED STUDY; DRUG EFFECT; ENDOTHELIUM CELL; HUMAN; HUMAN CELL; INFLAMMATION; MOUSE; NONHUMAN; PRIORITY JOURNAL; UMBILICAL VEIN;

ANGIOGENESIS INHIBITORS; CELLS, CULTURED; ENDOTHELIUM, VASCULAR; HUMANS; STILBENES; TUMOR NECROSIS FACTOR-ALPHA; UMBILICAL VEINS;

EID: 0035669040     PISSN: 00405957     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Conference Paper

References (14)

1. Cancer chemopreventive activity of resveratrol, a natural product derived from grapes
2. Resveratrol inhibits cyclooxygenase-2 transcription and activity in phorbol ester-treated human mammary epithelial cells
3. Resveratrol suppressed TNF-induced activation of nuclear transcription factors NF-kappa B, activation protein-1, and apoptosis: Potential role of reactive oxygen intermediates and lipid peroxidation
4. Suppression of nitric oxide synthase and the down-regulation of the activation of NFkappaB in macrophages by resveratrol
5. Activity in vitro of resveratrol on granulocyte and monocyte adhesion to endothelium
6. Adhesion receptors of the immune system
7. Cell adhesion in the immune system
8. Tumor necrosis factor α-induced vascular leakage involves PECAM-1 phosphorylation
9. Isolation of intact liver cells
10. Inhibition of human LDL oxidation by resveratrol
11. Antiplatelet activity of synthetic and natural resveratrol in red wine
12. TNFα induced EC F-actin depolymerization, new actin synthesis, and barrier dysfunction
13. Tyrosine kinase inhibition: An approach to drug development