General suppression of macrophage gene expression during Leishmania donovani infection

Journal of Immunology

Volumn 166, Issue 5, 2001, Pages 3416-3422

  Buates, S ^a   Matlashewski, G ^a  

^a MCGILL UNIVERSITY   (Canada)

Author keywords

[No Author keywords available]

Indexed keywords

COMPLEMENTARY DNA;

ANIMAL CELL; ARTICLE; CELL PROLIFERATION; CONTROLLED STUDY; FEMALE; GENE EXPRESSION; GENE INDUCTION; GENE REPRESSION; HOST PARASITE INTERACTION; LEISHMANIA DONOVANI; LEISHMANIASIS; MACROPHAGE; MOUSE; NONHUMAN; NUCLEOTIDE SEQUENCE; PARASITE DEVELOPMENT; PARASITE SURVIVAL; PHAGOSOME; PRIORITY JOURNAL; UNINDEXED SEQUENCE;

EID: 0035284822     PISSN: 00221767     EISSN: None     Source Type: Journal    
DOI: 10.4049/jimmunol.166.5.3416     Document Type: Article

References (25)

1. Intracellular survival of protozoan parasites with special reference to Leishmania spp., Toxoplasma gondii and Trypanosoma cruzi
2. Altered cell signaling and mononuclear phagocyte deactivation during intracellular infection
3. Deficient expression of co-stimulatory molecules on Leishmania-infected macrophages
4. Macrophage-T cell interaction in experimental visceral leishmaniasis: Failure to express costimulatory molecules on Leishmania-infected macrophages and its implication in the suppression of cell mediated immunity
5. B7-2 blockade enhances T cell responses to Leishmania donovani
6. Parasite accessory cell interactions in murine leishmaniasis. I. Evasion and stimulus-dependent suppression of the macrophage interleukin-1 response by Leishmania donovani
7. Modulation of in vitro monocyte cytokine responses to Leishmania donovani: Interferon-γ prevents parasite-induced inhibition of interleukin-1 production and primes monocytes to respond to Leishmania by producing both tumor necrosis factor-α and interleukin-1
8. c-fos and tumor necrosis factor gene expression in Leishmania donovani-infected macrophages
9. Alteration of Leishmania donovani infection levels by selective impairment of macrophage signal transduction
10. Leishmania donovani lipophosphoglycan selectively inhibits signal transduction in macrophages
11. Defective stimulus-response coupling in human monocytes infected with Leishmania donovani is associated with altered activation of protein kinase C
12. Inhibition of macrophage protein kinase C-mediated protein phosphorylation by Leishmania donovani lipophosphoglycan
13. Hybridization of denatured RNA and small DNA fragments transferred to nitrocellulose
14. Alteration of Leishmania donovani infection levels by selective impairment of macrophage signal transduction
15. Treatment of experimental leishmaniasis with the immunomodulators imiquimod and S-28463: Efficacy and mode of action
16. Intracellular infection by Leishmania donovani inhibits macrophage apoptosis
17. Leishmania donovani infection enhances macrophage viability in the absence of exogenous growth factor
18. CD40 ligand is required for protective cell-mediated immunity to Leishmania major
19. Disruption of CD40-CD40 ligand interactions results in an enhanced susceptibility to Leishmania amazonensis infection
20. Underproduction of interleukin-12 in susceptible mice during progressive leishmaniasis is due to decreased CD40 activity
21. Protective role of CD40 in Leishmania major infection at two distinct phases of cell-mediated immunity
22. Molecular cloning and functional expression of two monocyte chemoattractant protein I receptors reveals alternative splicing of the carboxyl-terminal tails
23. Impaired monocyte migration and reduced type 1 (Th1) cytokine responses in C-C chemokine receptor 2 knockout mice
24. Transbilayer inhibition of protein kinase C by the lipophosphoglycan from Leishmania donovani
25. The NF-κB and Iκ-B proteins: New discoveries and insights