Effect of sodium arsenite on iNOS expression and vascular hyporeactivity associated with cecal ligation and puncture in the rat

Shock

Volumn 15, Issue 1, 2001, Pages 73-76

  Hauser, Gabriel J ^a   Wasserloos, Karla ^b   Dayao, Emmanuel K ^a   Pitt, Bruce R ^b  

^a GEORGETOWN UNIVERSITY HOSPITAL   (United States)

^b UNIVERSITY OF PITTSBURGH SCHOOL OF MEDICINE   (United States)

Author keywords

Endotoxin; Heat shock; Norepinephrine; Sepsis; Stress response; TNF

Indexed keywords

ARSENITE SODIUM; ARSENOUS ACID DERIVATIVE; ENZYME INHIBITOR; HEAT SHOCK PROTEIN; INDUCIBLE NITRIC OXIDE SYNTHASE; NITRIC OXIDE SYNTHASE; NORADRENALIN; NOS2 PROTEIN, RAT; SODIUM DERIVATIVE; VASOCONSTRICTOR AGENT;

ANIMAL; ARTICLE; BLOOD VESSEL; CECUM; DRUG EFFECT; LIGATION; MALE; MESENTERY; METABOLISM; MORTALITY; PATHOPHYSIOLOGY; PHYSIOLOGY; PUNCTURE; RAT; SEPSIS; SURVIVAL RATE; VASOCONSTRICTION; WISTAR RAT;

ANIMALS; ARSENITES; BLOOD VESSELS; CECUM; ENZYME INHIBITORS; HEAT-SHOCK PROTEINS; LIGATION; MALE; MESENTERY; NITRIC OXIDE SYNTHASE; NITRIC OXIDE SYNTHASE TYPE II; NOREPINEPHRINE; PUNCTURES; RATS; RATS, WISTAR; SEPSIS; SODIUM COMPOUNDS; SURVIVAL RATE; VASOCONSTRICTION; VASOCONSTRICTOR AGENTS;

EID: 0035235207     PISSN: 10732322     EISSN: None     Source Type: Journal    
DOI: 10.1097/00024382-200115010-00012     Document Type: Article

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