Mitochondria deficient in complex I activity are depolarized by hydrogen peroxide in nerve terminals: Relevance to Parkinson's disease

Journal of Neurochemistry

Volumn 76, Issue 1, 2001, Pages 302-306

  Chinopoulos, Christos ^a   Adam Vizi, Vera ^a  

^a SEMMELWEIS UNIVERSITY   (Hungary)

Author keywords

Complex I; Hydrogen peroxide; Mitochondria; Mitochondrial membrane potential; Oxidative stress; Parkinson's disease

Indexed keywords

CARBOCYANINE; HYDROGEN PEROXIDE; IMIDAZOLE DERIVATIVE; IODINE DERIVATIVE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE DEHYDROGENASE (UBIQUINONE); ROTENONE;

ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; DEPOLARIZATION; ENERGY METABOLISM; ENZYME ACTIVITY; ENZYME INDUCTION; ENZYME MECHANISM; MEMBRANE POTENTIAL; MITOCHONDRIAL MEMBRANE; MITOCHONDRION; NERVE ENDING; NONHUMAN; OXIDATIVE STRESS; PARKINSON DISEASE; PATHOGENESIS; PRIORITY JOURNAL;

ANIMALS; CEREBRAL CORTEX; DOSE-RESPONSE RELATIONSHIP, DRUG; ELECTRON TRANSPORT COMPLEX I; ENERGY METABOLISM; GUINEA PIGS; HYDROGEN PEROXIDE; MITOCHONDRIA; NADH, NADPH OXIDOREDUCTASES; OXIDATIVE STRESS; PARKINSON DISEASE; PRESYNAPTIC TERMINALS; ROTENONE; SYNAPTOSOMES;

ANIMALIA;

EID: 0035162338     PISSN: 00223042     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1471-4159.2001.00060.x     Document Type: Article

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