PCR screening for common weak D types shows different distributions in three Central European populations

Transfusion

Volumn 41, Issue 1, 2001, Pages 45-52

  Müller, Thomas H ^{a,b,c,d,e,f,g}   Wagner, Franz F ^a,b,c,d,e,f   Trockenbacher, Andreas ^a,b,c,d,e,f   Eicher, Nicole I ^a,b,c,d,e,f   Flegel, Willy A ^a,b,c,d,e,f   Schönitzer, Diether ^a,b,c,d,e,f   Schunter, Friedrich ^a,b,c,d,e,f   Gassner, Christoph ^a,b,c,d,e,f  

^a Institute Springe   (Germany)

^b UNIVERSITY HOSPITAL ULM   (Germany)

^c German Red Cross Blood Donor Service Baden Württemberg   (Germany)

^d Central Laboratory in Bern   (Switzerland)

^e Central Institute for Blood Transfusion   (Austria)

^f Clinical Research   (United States)

^g BOEHRINGER INGELHEIM PHARMACEUTICALS INC   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

RHESUS D ANTIGEN;

ARTICLE; AUTOMATION; CAPILLARY ELECTROPHORESIS; DNA SEQUENCE; GEOGRAPHIC DISTRIBUTION; GERMANY; HUMAN; HUMAN CELL; HUMAN TISSUE; MAJOR CLINICAL STUDY; PHENOTYPE; POLYMERASE CHAIN REACTION; POPULATION RISK; RHESUS INCOMPATIBILITY; SEROTYPING;

ALLELES; AUSTRIA; DNA PRIMERS; GERMANY; HUMANS; ISOANTIGENS; PHENOTYPE; POLYMERASE CHAIN REACTION; RH-HR BLOOD-GROUP SYSTEM;

EID: 0035125518     PISSN: 00411132     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1537-2995.2001.41010045.x     Document Type: Article

References (19)

1. Molecular basis of weak D phenotypes
2. RHD/CE typing by polymerase chain reaction using sequence-specific primers
3. A simple method for high-quality frozen red cells in blood group serology
4. The human growth hormone locus: Nucleotide sequence, biology, and evolution
5. Weak D alleles express distinct phenotypes
6. Three molecular structures cause rhesus D category VI phenotypes with distinct immunohematologic features
7. Rh serology-coordinator's report
8. Rh phenotype prediction by DNA typing and its application to practice
9. Determination of autologous ABO, RHD, RHCE, KEL, JK and FY blood group genotypes by analysis of peripheral blood samples of patients who have recently received multiple transfusions
10. Genotyping of RHD by multiplex polymerase chain reaction analysis of six RHD-specific exons
11. Single-tube multiplex PCR-SSCP analysis distinguishes 7 common ABO alleles and readily identifies new alleles
12. Quantitation of Rh D antigen sites on weak D and D variant red cells by flow cytometry
13. Transcript analysis of D category phenotypes predicts hybrid Rh DCE-D proteins associated with alteratin of D epitopes
14. The genomic organization of the partial D category DVa: The presence of a new partial D associated with the DVa phenotype
15. The Rh blood group system: A review
16. Detection of Rh23 in the partial D phenotype associated with the D(Va) category
17. Frequencies of the blood groups ABO, Rhesus, D category VI, Kell, and of clinically relevant high-frequency antigens in southwestern Germany
18. The presence of an RHD pseudogene containing a 37 base pair duplication and a nonsense mutation in Africans with the Rh D-negative blood group phenotype
19. RHD gene deletion occured in the Rhesus box