Modified FGF4 signal peptide inhibits entry of herpes simplex virus type 1

Journal of Virology

Volumn 75, Issue 6, 2001, Pages 2634-2645

  Bultmann, H ^a   Busse, J S ^a   Brandt, C R ^a  

^a UNIVERSITY OF WISCONSIN MADISON   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

SIGNAL PEPTIDE;

ANIMAL CELL; ARTICLE; CELL ADHESION; CELL CULTURE; DRUG MECHANISM; ELECTRON MICROSCOPY; HERPES SIMPLEX VIRUS; NONHUMAN; PRIORITY JOURNAL; VIRION; VIRUS ADSORPTION; VIRUS INFECTIVITY; VIRUS INHIBITION;

ANIMALS; CERCOPITHECUS AETHIOPS; FIBROBLAST GROWTH FACTORS; HERPES SIMPLEX; HERPESVIRUS 1, HUMAN; PEPTIDES; PLAQUE ASSAY; PROTEIN SORTING SIGNALS; VERO CELLS; VIRION;

EID: 0035124125     PISSN: 0022538X     EISSN: None     Source Type: Journal    
DOI: 10.1128/JVI.75.6.2634-2645.2001     Document Type: Article

References (68)

1. A peptide nucleic acid (PNA) is more rapidly internalized in cultured neurons when coupled to a retro-inverso delivery peptide. The antisense activity depresses the target mRNA and protein in magnocellular oxytocin neurons
2. Evidence for an interaction of herpes simplex virus with chondroitin sulfate proteoglycans during infection
3. Magainins: A new family of membrane-active host defense peptides
4. Role of glycoprotein B of herpes simplex virus type I in viral entry and fusion
5. Glycoprotein C-dependent attachment of herpes simplex virus to susceptible cells leading to productive infection
6. Prediction of the secondary, structure of proteins from their amino acid sequence
7. Herpes simplex virus type 2 UL45 is a type II membrane protein
8. Fine mapping and molecular cloning of mutations in the herpes simplex virus DNA polymerase locus
9. The third helix of the antennapedia homeodomain translocates through biological membranes
10. Excretion of noninfectious virus particles lacking gH by a temperature.sensitive mutant of herpes simplex virus type I: Evidence that gH is essential for virion infectivity
11. Tat-mediated delivery of heterologous proleins into cells
12. HBTU activation for automated Fmoc solid-phase peptide synthesis
13. Anti-glyeoprotein D antibodies that permit adsorption but block infection by herpes simplex virus I prevent virion-cell fusion at the cell surface
14. Herpes simplex virus type I entry through a cascade of virus-cell interactions requires different roles of gD and gH in penetration
15. Entry of alphaherpesviruses mediated by poliovirus receptor related protein I and poliovirus receptor
16. Sequence and mapping analysis of the herpes simplex virus DNA polymerase gene predicts a c-terminal substrate binding domain
17. Herpes simplex virus stromal keratitis, is not titer-dependent and does not correlate with neurovirulence
18. The UL45 gene product is requires for herpes simplex virus type I glycoprotein B-induced fusion
19. Aphidicolin resistance in herpes simplex virus type 1 appears to alter substrate specificity in the DNA polymerase
20. Cross-linking of glycoprotein oligomers during herpes simplex virus type 1 entry
21. Glycoprotein C of herpes simplex virus type I plays a principal role in the adsorption of virus to cells and in infectivity
22. Glycoprotein C-independent binding of herpes simplex virus to cells requires cell surface heparan sulphate and glycoprotein B
23. Differences in the susceptibility of Herpes simplex virus types I and 2 to modified heparan compounds suggest serotype differences in viral entry
24. Neutralizing monoclonal antibodies specific for herpes simplex virus glycoprotein D inhibit virus penetration
25. A novel herpes simplex virus type I glycoprotein forms a complex with glycoprotein H (gH) and affects normal folding and surface expression of gH
26. Herpes simplex virus glycoprotein D mediates interference with herpes simplex virus infection
27. Potent suppression of HIV-1 replication in humans by T-20. A peptide inhibitor of gp41-mediated virus entry
28. The herpes simplex virus type 1 DNA polymerase gene: Site of phosphonoacetic acid resistance mutation in strain Angelotti is highly conserved
29. Herpes simplex virus glycoprotein D can bind to poliovirus receptor-related protein 1 or herpesvirus entry mediator, two structurally unrelated mediators of virus entry
30. Heparan sulfate proteoglycan binding by herpes simplex virus type I glycoproteins B and C, which differ in their contributions to virus attachment, penetration, and cell to cell spread
31. A herpes simplex virus mutant in which glycoprotein D sequences are replaced by β-galactosidase sequences binds to, but is unable to penetrate into cells
32. Inhibition of nuclear translocation of transcription factor NF-κβ By a synthetic peptide containing a cell membrane-permeable motif and nuclear localization sequence
33. Binding of herpes simplex virus to cellular heparan sulfate, an initial step in the adsorption process
34. Cell fusion induced by herpes simplex virus is promoted and suppressed by different viral glycoproteins
35. The structure and function of the HSV DNA replication proteins: Defining novel antiviral targets
36. Solid phase synthesis without repetitive acidolysls: Preparation of leucyl-alanyl-glycyl-valine using 9-fluorenylmethyloxycarbonylamino acids
37. Solid phase peptide synthesis. 1. The synthesis of a tetrapeptide
38. An analysis of the biological properties of monoclonal antibodies against glycoprotein D of herpes simplex virus and identification of amino acid substitutions that confer resistance to neutralization
39. Herpes simplex virus-1 entry into cells mediated by a novel member of the TNF/NGF receptor family
40. Structure function analysis of soluble forms of herpes simplex virus glycoprotein D
41. Monoclonal antibodies to distinct sites on herpes simplex virus (HSV) glycoprotein D block HSV binding to HVEM
42. The anti-HIV pseudopeptide HB-19 forms a complex with the cell-surface-expressed nucleolin independent of heparan sulfate proteoglycans
43. Nonendocytic, amphipathicity dependent cellular uptake of helical model peptides
44. Extensive cellular uptake into endothelial cells of an amphipathic β-sheet forming peptide
45. Cell penetration by transportan
46. Determinants of human immunodeficiency virus type I resistance to gp41-derived inhibitory peptides
47. A mutant herpes simplex virus type 1 unable to express glycoprotein L cannot enter cells and its particles lack glycoprotein H
48. Homopolymer mutational hot spots mediate herpes simplex virus resistance to acyclovir
49. In vivo protein transduction: Delivery of a biologically active protein into the mouse
50. Infection of polarized MDCK cells with herpes simplex virus 1: Two asymetrically distributed cell receptors interact with different viral proteins
51. Cell surface receptors for herpes simplex virus are heparan sulfate proteoglycans
52. Herpes virus-induced cell fusion that is dependent on cell surface heparan sulfate or soluble heparan
53. Entry of alphaherpesviruses into cells
54. Antiviral effects of apolipoprotein A-I and its synthetic amphipathic peptide analogs
55. Membrane interactions of synthetic peptides corresponding to amphipathic helical segments of the human immunodeficiency virus type-1 envelope glycoprotein
56. Interaction of herpes simplex virus glycoprotein C with mammalian cell surface molecules
57. Intraneuronal delivery, of protein kinase C pseudosubstrate leads to growth cone collapse
58. Glycoproteins gB. gD. and gHgL of herpes simplex virus type I are necessary and sufficient to mediate membrane fusion in a Cos celt transfection system
59. The HSV-I UL45 18 kDa gene product is a true late protein and a component of the virion
60. A truncated HIV-I tat protein basic domain rapidly translocates through the plasma membrane and accumulates in the celt nucleus
61. Glycoprotein H of herpes simplex virus type 1 requires glycoprotein L for transport to the surfaces of insect celts
62. Glycoprotein D of herpes simplex virus (HSV) binds directly to HVEM, a member of the tumor necrosis factor receptor superfamily and a mediator of HSV entry
63. Membrane fusion
64. Epidemiology of herpes simplex viruses
65. A synthetic peptide inhibitor of human immunodeficiency virus replication: Correlation between solution structure and viral inhibition
66. Initial interaction of herpes simplex virus with cells is binding to heparan sulfate
67. Peptides corresponding to the heptad repeat sequence of human parainfluenza virus fusion protein are potent inhibitors of virus infection