HMGI-C gene expression is not required for in vivo thyroid cell transformation

Carcinogenesis

Volumn 22, Issue 2, 2001, Pages 251-256

  Scala, Stefania ^a   Portella, Giuseppe ^a   Vitagliano, Donatella ^a   Ledent, Catherine ^b   Chiapetta, Gennaro ^c   Giancotti, Vincenzo ^d   Dumont, Jacques ^b   Fusco, Alfredo ^e  

^a UNIVERSITY OF NAPLES FEDERICO II   (Italy)

^b UNIVERSITÉ LIBRE DE BRUXELLES   (Belgium)

^c NATIONAL CANCER INSTITUTE   (Italy)

^d UNIVERSITY OF TRIESTE   (Italy)

^e UNIVERSITY MAGNA GRAECIA OF CATANZARO   (Italy)

Author keywords

[No Author keywords available]

Indexed keywords

HIGH MOBILITY GROUP PROTEIN; RADIOACTIVE IODINE; THYROGLOBULIN;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CARCINOGENESIS; CELL TRANSFORMATION; CONTROLLED STUDY; GENE DISRUPTION; GENE EXPRESSION; IN VIVO STUDY; MALIGNANT TRANSFORMATION; MOUSE; NONHUMAN; PAPILLOMA VIRUS; PRIORITY JOURNAL; PROMOTER REGION; THYROID CARCINOMA; THYROID CELL; TRANSCRIPTION REGULATION; TRANSGENIC MOUSE; VIRUS ONCOGENE;

EID: 0035109431     PISSN: 01433334     EISSN: None     Source Type: Journal    
DOI: 10.1093/carcin/22.2.251     Document Type: Article

References (26)

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4. Virus induction of human IFN beta gene expression requires the assembly of an enhanceosome
5. Developmental genetics. Living with bad architecture
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13. Increased expression of high mobility group protein I (Y) in high grade prostate cancer determined by in situ hybridization
14. The expression of the high mobility group HMGI(Y) proteins correlates with the malignant phenotype of human thyroid neoplasms
15. High mobility group protein I (Y) as potential tumor marker in human colorectal carcinomas
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