Low-dose vasopressin for reversing vasodilation during septic shock

Critical Care Medicine

Volumn 29, Issue 3, 2001, Pages 673-675

  Gazmuri, R J ^a   Shakeri, S A ^a  

^a VETERANS AFFAIRS MEDICAL CENTER   (United States)

Author keywords

Septic; Shock; Vasoconstrictor agents; Vasodilation; Vasopressins

Indexed keywords

VASOCONSTRICTOR AGENT; VASOPRESSIN;

DRUG EFFECT; EDITORIAL; HORMONE ACTION; HUMAN; HYPOTENSION; MORTALITY; PRESSOR RESPONSE; PRIORITY JOURNAL; SEPTIC SHOCK; VASOCONSTRICTION; VASODILATATION;

EID: 0035100663     PISSN: 00903493     EISSN: None     Source Type: Journal    
DOI: 10.1097/00003246-200103000-00042     Document Type: Editorial

References (10)

1. Induction of nitric oxide synthase and dual effects of nitric oxide and cyclooxygenase products in regulation of arterial contraction in human septic shock
2. The ATP-sensitive K+ channel mediates hypotension in endotoxemia and hypoxic lactic acidosis in dog
3. Vasopressin deficiency contributes to the vasodilation of septic shock
4. Vasopressin pressor hypersensitivity in vasodilatory septic shock
5. Low-dose vasopressin in the treatment of vasodilatory septic shock
6. Role of V1 receptors in the action of vasopressin on the baroreflex control of heart rate
7. Hemodynamic and metabolic effects of low-dose vasopressin infusions in vasodilatory septic shock
8. Endogenous vasopressin increases acute endotoxin shock-provoked gastrointestinal mucosal injury in the rat
9. The effects of vasopressin on endotoxin-induced attenuation of contractile responses in human gastroepiploic arteries in vitro
10. Physiologic concentrations of arginine vasopressin activate human platelets in vitro