Polymorphisms of the glucose transporter (GLUT1) gene are associated with diabetic nephropathy

Kidney International

Volumn 59, Issue 3, 2001, Pages 985-989

  Hodgkinson, A D ^a   Millward, B A ^a   Demaine, A G ^a  

^a PLYMOUTH UNIVERSITY PENINSULA SCHOOLS OF MEDICINE AND DENTISTRY   (United Kingdom)

Author keywords

Aldose reductase gene; ALR2; Diabetes mellitus; Heredity; Polymorphism of GLUT1

Indexed keywords

ALDEHYDE REDUCTASE; DNA; GLUCOSE TRANSPORTER;

ALLELE; ARTICLE; CAUCASIAN; CONTROLLED STUDY; DIABETIC NEPHROPATHY; DIABETIC RETINOPATHY; DISEASE DURATION; DNA POLYMORPHISM; FEMALE; GEL ELECTROPHORESIS; GENE AMPLIFICATION; GENETIC SUSCEPTIBILITY; GENOTYPE; HUMAN; HUMAN TISSUE; INSULIN DEPENDENT DIABETES MELLITUS; INTRON; MAJOR CLINICAL STUDY; MALE; PRIORITY JOURNAL; PROTEINURIA;

EID: 0035091486     PISSN: 00852538     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1523-1755.2001.059003985.x     Document Type: Article

References (15)

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6. Polymorphism in the 5′-end of the aldose reductase gene is strongly associated with the development of diabetic nephropathy in type 1 diabetes
7. Z-2 microsatellite allele is linked to increased expression of the aldose reductase gene in diabetic nephropathy
8. Aldose reductase gene polymorphisms and susceptibility to diabetic nephropathy in type 1 diabetes mellitus
9. Immunogold localisation of high-affinity glucose transporter isoform in normal rat kidney
10. d-glucose stimulates mesangial cell GLUT1 expression and basal and IGF-1 sensitive glucose uptake in rat mesangial cells: Implications for diabetic nephropathy
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13. The data and statistical analysis
14. +-coupled and facilitative transporters
15. Overexpression of glucose transporters in rat mesangial cells cultured in a normal glucose milieu mimics the diabetic phenotype