Loss of chromosome 16q in lobular carcinoma in situ

Human Pathology

Volumn 32, Issue 3, 2001, Pages 292-296

  Etzell, Joan E ^a   Devries, Sandy ^a   Chew, K ^a   Florendo, C ^a   Molinaro, A ^a   Ljung, B M ^a   Waldman, Frederic M ^b  

^a UNIVERSITY OF CALIFORNIA   (United States)

^b UNIVERSITY OF CALIFORNIA   (United States)

Author keywords

Breast neoplasms; Chromosome 16; Comparative genomic hybridization; Lobular carcinoma in situ

Indexed keywords

UVOMORULIN;

ARTICLE; BREAST CARCINOMA; CANCER RISK; CARCINOMA IN SITU; CHROMOSOME 12Q; CHROMOSOME 16Q; CHROMOSOME 17P; CHROMOSOME 1Q; CHROMOSOME 8P; CHROMOSOME LOSS; CLINICAL ARTICLE; COMPARATIVE GENOMIC HYBRIDIZATION; GENE EXPRESSION; HUMAN; HUMAN TISSUE; IMMUNOHISTOCHEMISTRY;

EID: 0035083435     PISSN: 00468177     EISSN: None     Source Type: Journal    
DOI: 10.1053/hupa.2001.22759     Document Type: Article

References (31)

1. Breast reductions: What to do with all the tissue specimens?
2. In situ breast carcinoma
3. Precancerous and cancerous breast lesions during lifetime and at autopsy: A study of 83 women
4. Lobular carcinoma in situ. A rare form of mammary cancer
5. A histologic classification of carcinoma in the breast
6. Simultaneous loss of E-cadherin and catenins in invasive lobular breast cancer and lobular carcinoma in situ
7. E-cadherin is a tumour/invasion suppressor gene mutated in human lobular breast cancers
8. E-cadherin inactivation in lobular carcinoma in situ of the breast: An early event in tumorigenesis
9. Lobular carcinoma in situ of the breast
10. Genetic alterations in lobular breast cancer by comparative genomic hybridization
11. Chromosomal alterations in ulcerative colitis-related neoplastic progression
12. Analysis of changes in DNA sequence copy number by comparative genomic hybridization in archival paraffin-embedded tumor samples
13. Computer image analysis of comparative genomic hybridization
14. C-erbB-2 oncogene protein in situ and invasive lobular breast neoplasia
15. c-erbB-2 overexpression and histological type of in situ and invasive breast carcinoma
16. Nuclear p53 protein accumulates preferentially in medullary and high-grade ductal but rarely in lobular breast carcinomas
17. p53 mutations and histological type of invasive breast carcinoma
18. Frequent overexpression of the cyclin D1 oncogene in invasive lobular carcinoma of the breast
19. Comparative allelotype of in situ and invasive human breast cancer: High frequency of microsatellite instability in lobular breast carcinomas
20. Point mutation of the E-cadherin gene in invasive lobular carcinoma of the breast
21. E-cadherin is inactivated in a majority of invasive human lobular breast cancers by truncation mutations through its extracellular domain
22. Genetic changes in intraductal breast cancer detected by comparative genomic hybridization
23. Comparative genomic hybridisation of ductal carcinoma in situ of the breast: Identification of regions of DNA amplification and deletion in common with invasive breast carcinoma
24. Analysis of loss of heterozygosity in 399 premalignant breast lesions at 15 genetic loci
25. Genomic alterations in tubular breast carcinomas
26. Allelotyping in ductal carcinoma in situ of the breast: Deletion of loci on 8p, 13q 16q, 17p, and 17q
27. Chromosomal alterations in ductal carcinomas in situ and their in situ recurrences
28. Comparative genomic hybridization analysis of lobular carcinoma in situ and atypical lobular hyperplasia and potential roles for gains and losses of genetic material in breast neoplasia
29. Mechanisms of inactivation of E-cadherin in breast cancer cell lines
30. Lobular neoplasia. Long term risk of breast cancer and relation to other factors