Pemphigus: Is there another half of the story?

Journal of Investigative Dermatology

Volumn 116, Issue 4, 2001, Pages 489-490

  Stanley, John R ^a   Nishikawa, Takeji ^a   Diaz, Luis A ^a   Amagai, Masayuki ^a  

^a Department of Dermatology ^*  (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CHOLINERGIC RECEPTOR; DESMOGLEIN; PROTEIN ANTIBODY;

BLISTER; EDITORIAL; HUMAN; MOUSE; NONHUMAN; PATHOPHYSIOLOGY; PEMPHIGUS; PRIORITY JOURNAL; SKIN DEFECT;

EID: 0035054272     PISSN: 0022202X     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1523-1747.2001.01307.x     Document Type: Editorial

References (21)

1. Autoantibodies against a novel epithelial cadherin in pemphigus vulgaris, a disease of cell adhesion
2. Absorption of pathogenic autoantibodies by the extracellular domain of pemphigus vulgaris antigen (Dsg3) produced by baculovirus
3. Pemphigus vulgaris antigen (Desmoglein 3) is localized in the lower epidermis, the site of blister formation in patients
4. The clinical phenotype of pemphigus is defined by the anti-desmoglein autoantibody profile
5. Toxin in bullous impetigo and staphylococcal scalded-skin syndrome targets desmoglein 1
6. Use of autoantigen-knockout mice in developing an active autoimmune disease model for pemphigus
7. Mucosal and mucocutaneous (generalized) pemphigus vulgaris show distinct autoantibody profiles
8. The anti-desmoglein 1 autoantibodies in pemphigus vulgaris sera are pathogenic
9. Identification of pemphigus vulgaris antigen extracted from normal human epidermis and comparison with pemphigus foliaceus antigen
10. Detection of pemphigus vulgaris and pemphigus foliaceus antigens by immunoblot analysis using different antigen sources
11. Characterization of autoantibodies in pemphigus using antigen-specific enzyme-linked immunosorbent assays with baculovirus-expressed recombinant desmogleins
12. Targeted disruption of the pemphigus vulgaris antigen (desmoglein 3) gene in mice causes loss of keratinocyte cell adhesion with a phenotype similar to pemphigus vulgaris
13. Pemphigus sera recognize conformationally sensitive epitopes in the amino-terminal region of desmoglein-1 (Dsg1)
14. Desmoglein 3-ELISA. A pemphigus vulgaris-specific diagnostic tool
15. Explanation for the clinical and microscopic localization of lesions in pemphigus foliaceus and vulgaris
16. Late development of antidesmoglein 1 antibodies in pemphigus vulgaris: Correlation with disease progression
17. The pathophysiologic significance of nondesmoglein targets of pemphigus autoimmunity. Development of antibodies against keratinocyte cholinergic receptors in patients with pemphigus vulgaris and pemphigus foliaceus
18. Pemphigus vulgaris antibody identifies pemphaxin
19. Antibodies against keratinocyte antigens other than desmoglein 1 and 3 can induce pemphigus vulgaris-like lesions
20. Pemphigus foliaceus sera recognize an N-terminal fragment of bovine desmoglein 1
21. A monoclonal antibody to the desmosomal glycoprotein desmoglein I binds the same polypeptide as human autoantibodies in pemphigus foliaceus