The Fas death pathway as a mechanism of multiple organ dysfunction syndrome: Not so fast

Critical Care Medicine

Volumn 29, Issue 4, 2001, Pages 901-902

  Hotchkiss, R S ^a   Karl, I E ^a  

^a WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Apoptosis; Cell death; Lymphocytes

Indexed keywords

FAS ANTIGEN;

CELL KILLING; CELLULAR IMMUNITY; EDITORIAL; EFFECTOR CELL; IMMUNE RESPONSE; MULTIPLE ORGAN FAILURE; PRIORITY JOURNAL;

EID: 0035046574     PISSN: 00903493     EISSN: None     Source Type: Journal    
DOI: 10.1097/00003246-200104000-00046     Document Type: Editorial

References (9)

1. The Fas death factor
2. Lymphocyte survival: The struggle against death
3. Expression of Fas (CD95) and Fas ligand on peripheral blood mononuclear cells in critical illness and association with multiorgan dysfunction severity and survival
4. Cross-talk in cell death signaling
5. Increased mucosal B-lymphocyte apoptosis during polymicrobial sepsis is a Fas ligand but not an endotoxin-mediated process
6. p53-dependent and -independent pathways of apoptotic cell death in sepsis
7. Apoptotic cell death in patients with sepsis, shock, and multiple organ dysfunction
8. CD95/CD95 ligand interactions on epithelial cells in host defense to Psuedomonas aeruginosa
9. Fas-FasL interactions modulate host defense against systemic Candida albicans infection