Neuroglial and its roles in pathogenesis of the Alzheimer's disease

Chinese Pharmaceutical Journal

Volumn 36, Issue 4, 2001, Pages 217-220

  Feng, Z ^a   Zhang, J T ^a  

^a NONE

Author keywords

[No Author keywords available]

Indexed keywords

AMYLOID BETA PROTEIN; APOLIPOPROTEIN E; CD45 ANTIGEN; COMPLEMENT MEMBRANE ATTACK COMPLEX; COMPLEMENT RECEPTOR; GLIAL FIBRILLARY ACIDIC PROTEIN; INTERLEUKIN 3; LIPOPOLYSACCHARIDE; MAJOR HISTOCOMPATIBILITY ANTIGEN CLASS 1; MAJOR HISTOCOMPATIBILITY ANTIGEN CLASS 2; NERVE GROWTH FACTOR;

ALZHEIMER DISEASE; ARTICLE; CENTRAL NERVOUS SYSTEM; GLIA; HUMAN; NEUROFIBRILLARY TANGLE; PATHOGENESIS;

EID: 0035038679     PISSN: 10012494     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (18)

1. -2 forming NADPH oxidase in microglia, monocytes, and neutrophils. A possible inflammatory mechanism of neuronal damage in Alzheimer' s disease[J]
2. The microglial cell. A historical review[J]
3. Complement component C1q modulates the phagocytosis of Aβ by microglia[J]
4. Progression of clinical deterioration and pathological changes in patients with Alzheimer disease evaluated at biopsy and autopsy[J]
5. Mechanisms of cell death in Alzheimer disease-immunopathology[J]
6. Support of homeostatic glial cell signaling: A novel therapeutic approach by propentofylline [J]
7. +-dependent glutamate uptake[J]
8. Microglial activation in Alzheimer disease: Association with APOE genotype [J]
9. NGF induces apoptosis in a human neuroblastoma cell line expressing the neurotrophin receptor p75NTR[J]
10. Neuroimmune mechanisms in Alzheimer, s disease pathogenesis [J]
11. Translation of the Alzheimer amyloid precursor protein mRNA is up-regulated by interleukin-1 through 5′-untranslated region sequences[J]
12. Amylold-β induces chemokine secretion and monocyte migration across a human blood-brain barrier model [J]
13. Immunohistochemical study of the β-chemokine receptors CCR3 and CCR5 and their ligands in normal and Alzheimer's disease brains[J]
14. Chronic overproduction of transforming growth factor-β1 by astrocytes promotes Alzheimer's disease-like microvascular degeneration in transgenic mice [J]
15. Cytokines associated with amyloid plaques in Alzheimer's disease brain stimulate human glial and neuronal cell cultures to secrete early complement proteins, but not C1-inhibitor[J]
16. Temporal accrual of complement proteins in amyloid plaques in Down' s syndrome with Alzheimer' s disease[J]
17. Oncostatin M and the interleukin-6 and soluble interleukin-6 receptor complex regulate α1-antichymotrypsin expression in human cortical astrocytes [J]
18. Glial-derived proteins activate cultured astrocytes and enhance β amyloid-induced glial activation[J]