Tumor necrosis factor-dependent adhesions as a major protective mechanism early in septic peritonitis in mice

Infection and Immunity

Volumn 69, Issue 6, 2001, Pages 3550-3555

  Echtenacher, B ^a   Weigl, K ^a   Lehn, N ^a   Mannel D N ^a  

^a UNIVERSITY OF REGENSBURG   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

ANTIBIOTIC AGENT; CIPROFLOXACIN; HEPARIN; HIRUDIN; METRONIDAZOLE; PERITONEAL DIALYSIS FLUID; PROCOAGULANT; TUMOR NECROSIS FACTOR; UROKINASE;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BACTERIAL COUNT; BACTERIAL PERITONITIS; BLOOD CLOTTING; CORRELATION FUNCTION; IMMUNITY; KIDNEY; LETHALITY; LIVER; MALE; MORTALITY; MOUSE; NONHUMAN; PERITONEUM ADHESION; PRIORITY JOURNAL; SURGICAL PATIENT;

ADHESIONS; ANIMALS; CECUM; COLONY COUNT, MICROBIAL; DISEASE MODELS, ANIMAL; HEPARIN; HIRUDINS; IMMUNITY, NATURAL; LIGATION; MALE; MICE; PERITONITIS; SEPSIS; TUMOR NECROSIS FACTOR-ALPHA; URINARY PLASMINOGEN ACTIVATOR;

EID: 0035019731     PISSN: 00199567     EISSN: None     Source Type: Journal    
DOI: 10.1128/IAI.69.6.3550-3555.2001     Document Type: Article

References (30)

1. Divergent efficacy of antibody to tumor necrosis factor-alpha in intravascular and peritonitis models of sepsis
2. Local serum application: Restoration of sufficient host defense in human peritonitis
3. Modulators of coagulation. A critical appraisal of their role in sepsis
4. Combination of antibiotic treatment with the thrombin inhibitor recombinant hirudin for the therapy of experimental Klebsiella pneumoniae sepsis
5. Formation of fibrin monomers in experimental disseminated intravascular coagulation and its inhibition by recombinant hirudin
6. Tumor necrosis factor (TNF) stimulates plasminogen activator inhibitor (PAI) production by endothelial cells and decreases blood fibrinolytic activity in the rat
7. Requirement of endogenous tumor necrosis factor/cachectin for recovery from experimental peritonitis
8. Survival from cecal ligation and puncture and the formation of fibrous adhesions in the peritoneal cavity depend on endogenous tumor necrosis factor
9. Critical protective role of mast cells in a model of acute septic peritonitis
10. Anti-tumor necrosis factor antibody therapy fails to prevent lethality after cecal ligation and puncture or endotoxemia
11. Treatment of septic shock with the tumor necrosis factor receptor: Fc fusion protein
12. Cellular mechanism of intraabdominal abscess formation by Bacteroides fragilis
13. Mediators of septic shock: New approaches for interrupting the endogenous inflammatory cascade
14. Good and laudable pus
15. Tumor necrosis factor-alpha: A marker for peritoneal adhesion formation
16. Prognosis and treatment of peritonitis. Do we need new scoring systems?
17. On the binding of tumor necrosis factor (TNF) to heparin and the release in vivo of the TNF-binding protein I by heparin
18. Immunologic mast cell-mediated responses and histamine release are attenuated by heparin
19. The biology of peritonitis and implications for treatment
20. The pulmonary inflammatory response to experimental fecal peritonitis: Relative roles of tumor necrosis factor-alpha and endotoxin
21. Assessment of antibiotic-mediated endotoxin release
22. Heparin oligosaccharides bind L- and P-selectin and inhibit acute inflammation
23. Failure of prophylactic and therapeutic use of a murine anti-tumor necrosis factor monoclonal antibody in Escherichia coli sepsis in the rabbit
24. Multiple roles for IL-12 in a model of acute septic peritonitis
25. Therapeutic uses of heparin beyond its traditional role as an anticoagulant
26. Fibrinolytic response to tumor necrosis factor in healthy subjects
27. Characterization and fibrinolytic properties of human omental tissue mesothelial cells. Comparison with endothelial cells
28. Sepsis and septic shock - A review of laboratory models and a proposal
29. Management of secondary peritonitis
30. Anti-inflammatory therapies to treat sepsis and septic shock: A reassessment