Perinatal development influences mechanisms of bradykinin-induced relaxations in pulmonary resistance and conduit arteries differently

Cardiovascular Research

Volumn 51, Issue 1, 2001, Pages 140-150

  Boels, P J ^a   Deutsch, J ^a   Gao, B ^b   Haworth, S G ^a  

^a UNIVERSITY COLLEGE LONDON   (United Kingdom)

^b UNIVERSITY COLLEGE LONDON   (United Kingdom)

Author keywords

Developmental biology; Endothelial factors; Prostaglandins; Pulmonary circulation; Vasoconstriction dilation

Indexed keywords

ACETYLCHOLINE; BRADYKININ; INDOMETACIN; N(G) NITROARGININE METHYL ESTER; NITRIC OXIDE; POTASSIUM ION; PROADIFEN; PROSTAGLANDIN DERIVATIVE; SUBSTANCE P; VASODILATOR AGENT;

AGE; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTERY DILATATION; ARTICLE; CONTROLLED STUDY; DRUG SENSITIVITY; FETUS; HYPERPOLARIZATION; IN VITRO STUDY; LUNG BLOOD VESSEL; LUNG CIRCULATION; LUNG RESISTANCE; MEDIATOR RELEASE; NEWBORN; NONHUMAN; PERINATAL DEVELOPMENT; PRIORITY JOURNAL; PULMONARY ARTERY; SWINE;

ACETYLCHOLINE; ANALYSIS OF VARIANCE; ANIMALS; ANIMALS, NEWBORN; BRADYKININ; CYCLOOXYGENASE INHIBITORS; CYTOCHROME P-450 ENZYME SYSTEM; DOSE-RESPONSE RELATIONSHIP, DRUG; ENZYME INHIBITORS; INDOMETHACIN; NG-NITROARGININE METHYL ESTER; NITRIC OXIDE SYNTHASE; PROADIFEN; PULMONARY ARTERY; SUBSTANCE P; SWINE; VASCULAR RESISTANCE; VASODILATOR AGENTS;

EID: 0035009871     PISSN: 00086363     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0008-6363(01)00275-9     Document Type: Article

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