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Volumn 21, Issue 2 A, 2001, Pages 1207-1212

TGFβ1 induces caspase-dependent but death-receptor independent apoptosis in lymphoid cells

Author keywords

Apoptosis; Caspases; Death receptors; Lymphoma; Mitochondria; TGF 1

Indexed keywords

CASPASE; PROTEIN BAX; PROTEIN BCL 2; PROTEIN BCL X; TRANSFORMING GROWTH FACTOR BETA1;

EID: 0035002231     PISSN: 02507005     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article
Times cited : (19)

References (32)
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  • 18
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  • 23
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    • Mechanism that contribute to the development of lymphoid malignancies: Roles for genetic alterations and cytokine production
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    • Levy, L.S.1    Bost, K.L.2
  • 28
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    • Caspase cleaved BID targets mitochondria and is required for cytochrome c release, while BCL-XL prevents this release but not tumor necrosis factor R1/Fas death
    • (1999) J Biol Chem , vol.274 , pp. 1156-1163
    • Gross, A.1    Yin, X.M.2    Wang, K.3
  • 31
    • 0034284209 scopus 로고    scopus 로고
    • Allday apoptosis induced by TGF-β1 in Burkitt's lymphoma cells is caspase 8 dependent but is death receptor independent
    • (2000) J Immunol , vol.165 , pp. 2500-2510
    • Gareth, J.1    Inman2    Martin, J.3


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.