TNF-α inhibits 3T3-L1 adipocyte differentiation without downregulating the expression of C/EBPβ and δ

Endocrine Journal

Volumn 48, Issue 2, 2001, Pages 249-253

  Kurebayashi, Shogo ^a   Sumitani, Satoru ^a   Kasayama, Soji ^a   Jetten, Anton M ^b   Hirose, Takahisa ^c  

^a OSAKA UNIVERSITY MEDICAL SCHOOL   (Japan)

^b NATIONAL INSTITUTE OF ENVIRONMENTAL HEALTH SCIENCES   (United States)

^c Nishinomiya Municipal Central Hospital   (Japan)

Author keywords

Adipocyte differentiation; C EBP , , ; PPAR ; TNF

Indexed keywords

CCAAT ENHANCER BINDING PROTEIN; CCAAT ENHANCER BINDING PROTEIN BETA; CCAAT ENHANCER BINDING PROTEIN SIGMA; TUMOR NECROSIS FACTOR ALPHA; UNCLASSIFIED DRUG;

ADIPOCYTE; ANIMAL CELL; ARTICLE; CELL DIFFERENTIATION; CELL STRAIN 3T3; CONTROLLED STUDY; DOWN REGULATION; GENE EXPRESSION; GENETIC ANALYSIS; MOUSE; NONHUMAN; PROTEIN BINDING; PROTEIN EXPRESSION; STIMULUS RESPONSE;

EID: 0034981551     PISSN: 09188959     EISSN: None     Source Type: Journal    
DOI: 10.1507/endocrj.48.249     Document Type: Article

References (20)

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5. CCAAT/enhancer-binding protein α is sufficient to initiate the 3T3-L1 adipocyte differentiation program
6. Stimulation of adipogenesis in fibroblasts by PPARγ2, a lipid-activated transcription factor
7. Conditional ectopic expression of C/EBPβ in NIH-3T3 cells induces PPARγ and stimulates adipogenesis
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11. Transcriptional repression of the C/EBPα and GLUT4 genes in 3T3-L1 adipocytes by tumor necrosis factor α regulations is coordinate and independent of protein synthesis
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15. Retinoid-related orphan receptor γ is essential for lymphoid organogenesis and controls apoptosis during thymopoiesis
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