Possible mechanisms to explain the absence of hyperkalaemia in addison's disease

Nephrology Dialysis Transplantation

Volumn 16, Issue 6, 2001, Pages 1280-1284

  Gagnon, Raymonde F ^a   Halperin, Mitchell L ^b  

^a MONTREAL GENERAL HOSPITAL   (Canada)

^b ST MICHAEL'S HOSPITAL   (Canada)

Author keywords

Adrenal insufficiency; Aldosterone; Anion gap; Cortical collecting duct; Hyponatraemia; Potassium

Indexed keywords

ALBUMIN; BICARBONATE; CHLORIDE; CREATININE; FLUDROCORTISONE; GLUCOSE; HEMOGLOBIN; HYDROCORTISONE; HYDROCORTISONE SODIUM SUCCINATE; POTASSIUM; SODIUM; SODIUM CHLORIDE; VASOPRESSIN;

ADDISON DISEASE; ADULT; ALBUMIN BLOOD LEVEL; ANION GAP; ARTICLE; BICARBONATE BLOOD LEVEL; BLOOD LEVEL; CASE REPORT; CHLORIDE BLOOD LEVEL; CHLORIDE URINE LEVEL; CONTROLLED STUDY; CREATININE BLOOD LEVEL; CREATININE URINE LEVEL; GLUCOSE BLOOD LEVEL; GLUCOSE URINE LEVEL; HUMAN; HYPERKALEMIA; HYPONATREMIA; INAPPROPRIATE VASOPRESSIN SECRETION; MALE; OSMOLALITY; POTASSIUM BLOOD LEVEL; POTASSIUM URINE LEVEL; PRIORITY JOURNAL; SODIUM BLOOD LEVEL; SODIUM URINE LEVEL; UREA NITROGEN BLOOD LEVEL; URINE LEVEL;

EID: 0034970312     PISSN: 09310509     EISSN: None     Source Type: Journal    
DOI: 10.1093/ndt/16.6.1280     Document Type: Article

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