Effects of TH-2 type cytokines on human airway epithelial cells: Interleukins-4, -5, and -13

Cell Biology International

Volumn 25, Issue 6, 2001, Pages 563-566

  Relova, A J ^a   Kampf, C ^a   Roomans, G M ^a  

^a UPPSALA UNIVERSITY   (Sweden)

Author keywords

Airway epithelium; Asthma; Cell proliferation; Cytokines; Inflammation

Indexed keywords

CYTOKINE; INTERLEUKIN 13; INTERLEUKIN 4; INTERLEUKIN 5;

ARTICLE; ASTHMA; CELL DEATH; CELL PROLIFERATION; CONTROLLED STUDY; HELPER CELL; HUMAN; HUMAN CELL; PATHOGENESIS; RESPIRATORY EPITHELIUM; TH2 CELL;

EID: 0034952774     PISSN: 10656995     EISSN: None     Source Type: Journal    
DOI: 10.1006/cbir.2000.0687     Document Type: Article

References (11)

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4. Effects of TNF-alpha, IFN-gamma and- IL-beta on normal human bronchial epithelial cells
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7. IL-4 and insulin-like growth factor-I inhibit the decline in Bcl-2 and promote the survival of IL-3-deprived myeloid progenitors
8. Rapid colorimetric assay for cellular growth and survival: Application to proliferation and cytotoxicity assays
9. Proliferation of the BCL1 B cell lymphoma induced by IL-4 and IL-5 is dependent on IL-6 and GM-CSF
10. IL-13 induces proliferation, Ig isotype switching, and Ig synthesis by immature human fetal B cells
11. Objective quantitative analysis of eosinophils and bronchial epithelial cells in induced sputum by laser scanning cytometry