Effectiveness of acid suppression in preventing gastroesophageal reflux disease (GERD) after successful treatment of Helicobacter pylori infection

Digestive Diseases and Sciences

Volumn 46, Issue 7, 2001, Pages 1567-1572

  Rokkas, T ^a,b   Ladas, S D ^b   Liatsos, C ^a   Panagou, E ^a   Karameris, A ^a   Raptis, S A ^b  

^a 401 GENERAL MILITARY HOSPITAL OF ATHENS   (Greece)

^b UNIVERSITY OF ATHENS   (Greece)

Author keywords

Acid suppression; Eradication; Esophagitis; GERD; Helicobacter pylori

Indexed keywords

OMEPRAZOLE;

ADULT; ARTICLE; CLINICAL TRIAL; CONTROLLED CLINICAL TRIAL; CONTROLLED STUDY; DISEASE PREDISPOSITION; ERADICATION THERAPY; ESOPHAGITIS; FEMALE; FOLLOW UP; GASTROESOPHAGEAL REFLUX; GASTROINTESTINAL ENDOSCOPY; GRAM NEGATIVE INFECTION; HUMAN; MAJOR CLINICAL STUDY; MALE; PRIORITY JOURNAL; RANDOMIZED CONTROLLED TRIAL; SYMPTOMATOLOGY;

ADULT; ANTI-ULCER AGENTS; ENDOSCOPY, GASTROINTESTINAL; FEMALE; GASTRIC ACID; GASTROESOPHAGEAL REFLUX; HELICOBACTER INFECTIONS; HELICOBACTER PYLORI; HUMANS; MALE; MIDDLE AGED; OMEPRAZOLE; PROSPECTIVE STUDIES;

EID: 0034939760     PISSN: 01632116     EISSN: None     Source Type: Journal    
DOI: 10.1023/A:1010616710501     Document Type: Article

References (48)

1. Pyloric Campylobacter infection and gastroduodenal disease
2. Infection with Campylobacter pylori
3. Campylobacter pylori and peptic ulcer disease
4. Cure of duodenal ulcer associated with eradication of Helicobacter pylori
5. The role of infectious agents in peptic ulcer disease
6. Effect of treatment of Helicobacter pylori infection and the long term reccurence of gastric and duodenal ulcer; a randomized, controlled study
7. Maintenance treatment versus Helicobacter pylori eradication therapy in preventing rebleeding of peptic ulcer disease. A clinical trial and follow up for two years
8. Treatment of Helicobacter pylori reduces the rate of rebleeding in peptic ulcer disease
9. Helicobacter pylori eradication reduces the rate of rebleeding in ulcer haemorrhage
10. Eradication of Helicobacter pylori, reduces the possibility of rebleeding in peptic ulcer disease
11. The seroprevalence of cagA-positive Helicobacter pylori strains in the spectrum of gastroesophageal reflux disease
12. Helicobacter pylori and gastroesophageal reflux disease
13. Helicobacter pylori is not a pathogenetic factor in GERD or its complications
14. Evaluation of Helicobacter pylori in reflux oesophagitis and Barrett's oesophagus
15. Decreased prevalence of H. pylori infection in Gastroesophageal reflux disease
16. Curing Helicobacter pylori infection in patients with duodenal ulcer may provoke reflux esophagitis
17. Classification and grading of gastritis. The updated Sydney system
18. The endoscopic assessment of of esophagitis: A progress report on observer agreement
19. Prevalence and clinical spectrum of gastroesophageal reflux disease: A population-based study in Olmsted County, Minnesota
20. Gastric acid secretion in refractory gastroesophageal reflux disease
21. Reflux esophagitis in patients with Zollinger-Ellison syndrome
22. An inverse relation between cagA+ strains of Helicobacterr pylori infection and risk of esophageal and gastric cardia adenocarcinoma
23. The incidence of reflux esophagitis. A study on endoscopy report from a defined catchment area in Sweden
24. The prevalence of gastro-esophageal reflux disease
25. Urea hydrolysis in Helicobacter pylori infection
26. H. pylori related increase in omeprazole (OME) effect is associated with ammonia production
27. Production of volatile amines by Helicobacter pylori (Hp) is not relevant in vivo
28. The pathophysiology of gastrooesophageal reflux disease: An overview
29. Gastric mucosal damage and adaptive protection by ammonia and ammonium ions in rats
30. Helicobacter pylori infection and abnormalities of acid secretion in patients with duodenal ulcer disease
31. Eradication of Helicobacter pylori infection lowers gastrin mediated acid secretion by two thirds in patients with duodenal ulcer
32. Eradication of Helicobacter pylori in patients with duodenal ulcer lowers basal and peak acid outputs to gastrin releasing peptide and pentagastrin
33. Basal and stimulated gastrin levels and gastric acid output five months after therapy for Helicobacter pylori eradication in duodenal ulcer patients
34. Helicobacter pylori infection and chronic gastric acid hypersecretion
35. Eradication of Helicobacter pylori, increases gastric acidity in patients with atrophic gastritis of the corpus-evaluation of 24-h pH monitoring
36. +-ATPase
37. Effect of a Campylobacter pylori protein on acid secretion by parietal cells
38. Relationship between severity of H. pylori (HP) gastritis and gastric acid-pepsin secretion in man
39. Improved fold width and increased acid secretion after eradication of the organism in Helicobacter pylori associated enlarged fold gastritis
40. Nitric oxide mediates the inhibition by interleukin-1 beta of pentagastrin-stimulated rat gastric acid secretion
41. Helicobacter pylori and gastric acid: Biological and therapeutic implications
42. Muscarinic receptors in gastric mucosa are increased in peptic ulcer disease
43. 2 synthesis by the stomach, and retards gastric emptying
44. g-nitro-L-arginine methyl ester on duodenal alkaline secretory and ulcerogenic responses induced by mepirizole in rats
45. Serologic detection of CagA Helicobacter pylori strains predicts the presence of peptic ulcer in young dyspeptic patients
46. Induction of various cytokines and development of severe mucosal inflammation by cagA gene positive Helicobacter pylori, strains
47. Opposing time trends of peptic ulcer and reflux disease