Potent inactivation of representative members of each PKC isozyme subfamily and PKD via S-thiolation by the tumor-promotion/progression antagonist glutathione but not by its precursor cysteine

Carcinogenesis

Volumn 22, Issue 8, 2001, Pages 1221-1229

  Chu, F ^a   Ward, N E ^a   O'Brian, C A ^a  

^a UNIVERSITY OF TEXAS   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CYSTEINE; DIAMIDE; DISULFIDE; GLUTATHIONE; OXIDIZING AGENT; PROTEIN KINASE; PROTEIN KINASE C; PROTEIN KINASE C ALPHA; PROTEIN KINASE C BETA1; PROTEIN KINASE C BETA2; PROTEIN KINASE C DELTA; PROTEIN KINASE C EPSILON; PROTEIN KINASE C GAMMA; PROTEIN KINASE C ZETA; PROTEIN KINASE D; UNCLASSIFIED DRUG;

ARTICLE; CONCENTRATION RESPONSE; CONTROLLED STUDY; DISULFIDE BOND; ENZYME INACTIVATION; PRIORITY JOURNAL; STOICHIOMETRY; TUMOR GROWTH; TUMOR PROMOTION;

EID: 0034903753     PISSN: 01433334     EISSN: None     Source Type: Journal    
DOI: 10.1093/carcin/22.8.1221     Document Type: Article

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