Clinical evaluation of pseudo-ceramide containing cream for the patients with atopic dermatitis

Nishinihon Journal of Dermatology

Volumn 63, Issue 4, 2001, Pages 457-461

  Mizutani, H ^a  

^a NONE

Author keywords

[No Author keywords available]

Indexed keywords

AMIDASE; CERAMIDE; SPHINGOMYELIN;

ARTICLE; ATOPIC DERMATITIS; CLINICAL ARTICLE; CLINICAL TRIAL; CREAM; DERMATITIS; DERMATOPHAGOIDES PTERONYSSINUS; ENZYME DEFICIENCY; FEMALE; HUMAN; MALE; SKIN WATER LOSS; TREATMENT OUTCOME;

EID: 0034870956     PISSN: 03869784     EISSN: None     Source Type: Journal    
DOI: 10.2336/nishinihonhifu.63.457     Document Type: Article

References (15)

1. Epidermal lipids, barrier function, and desquamation
2. Decreased level of ceramides in stratum corneum of atopic dermatitis: An Etiologic factor in atopic dry skin?
3. Pseudo-acylceramide with linoleic acid produces selective recovery of diminished cutaneous barrier function in essential fatty acid deficient rats and has an inhibitory effect on epidermal hyperplasia
4. Percutaneous sensitization with mite antigens elicits Th 2-dominant cytokine response in a cutaneous barrier-disrupted model
5. Abnormal expression of sphingomyelin acylase in atopic dermatitis: An etiologic factor for ceramide deficiency?
6. High-expression of sphingomyelin deacylase is an important determinant of ceramide deficiency leading to barrier disruption in atopic dermatitis
7. The skin of atopic dermatitis patients contains a novel enzyme, glucosylceramide sphingomyelin deacylase, which cleaves the N-acyl linkage of sphingomyelin and glucosylceramide
8. Successful protection of antigen specific dermatitic reaction in atopic dermatitis by topical O-acylceramides
9. A speculative view of atopic dermatitis: Barrier dysfunction in pathogenesis
10. Skin moisturizers: Development and clinical use ceramides